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Saturday, January 19, 2019

Anyone concerned about Kaposi's sarcoma in Chronic Fatigue Syndrome should read this excerpt from The Chronic Fatigue Syndrome Epidemic Cover-up

     In the same issue, Neenyah Ostrom reported on a new develop-ment that should have given pause to anyone married to the HIV/AIDS paradigm. She wrote, “In a striking reversal of ‘AIDS’ dogma, two recent scientific reports presented data demonstrating that the Human Immunodeficiency virus (HIV) is not the cause of Kaposi’s sarcoma (KS). These reports, in the British medical journal The Lancet, suggest that the KS seen in gay men (and others) with ‘AIDS’ may be caused by an ‘as yet unidentified’ infectious, sexually transmitted agent.”

     In another ill-considered judgment, the AIDS establishment, rather than recognizing this development as an epidemiological warning from nature that HIV-negative KS meant that HIV could not be the cause of AIDS (since KS was considered one of the dramatic hallmarks of AIDS), decided to split HIV/AIDS and KS into separate epidemics rather than lose face with the public by admitting that they had gotten the basics of the epidemic dead wrong.

     Ostrom pointed out that neither of the new Lancet reports “takes into account research performed in 1985 by researchers at the University of Miami School of Medicine in which KS was found in greater than 94 percent of autopsied patients with ‘AIDS,’ leading those investigators to postulate that ‘this autopsy series suggests that Kaposi’s sarcoma may be present in all patients with AIDS.’ (L.B. Moskovitz et al., Human Pathology, May, 1985).”

     In their study, the University of Miami scientists stated that their “findings indicated that Kaposi’s sarcoma is more common and has a wider morphological spectrum in AIDS than is generally appre-ciated.”

     Ostrom also reported, “More than 90 percent of the patients studied by the group displayed ‘microscopic evidence of Kaposi’s sarcoma in one or more organs.’ In fact, these investigators found that ‘in only one of the patients was Kaposi’s sarcoma limited to the skin’; only 26 percent of the group had cutaneous (on the surface of the skin) KS. The most common sites of identification of KS were lymph nodes and spleen.”

     For anyone with half a brain, what the University of Miami research suggested was that KS was the fundamental pathological event in AIDS, not a secondary one, and the presence of it in people without HIV indicated that HIV could possibly be ruled out as the real cause of AIDS. One of the reasons this did not register with the scientific community was that rampant fear among doctors had resulted in a limited number of autopsies being performed on AIDS patients. More autopsies might have resulted in a shift in the whole HIV/AIDS paradigm.

     The University of Miami scientists stated, “The [94.2%] prevalence of Kaposi’s sarcoma in patients with AIDS that was observed in this series had not been reported previously; there are a number of possible explanations for this disparity.” Ostrom reported that one of the reasons for the discrepancies was “that the autopsies examined many organs not just skin and occasional lymph nodes. They note, however, that autopsy ‘is not an infallible method’ for identifying KS. For example, tissues can be altered beyond recognition by other infectious agents; inadequate sampling (of lymph node tissue, primarily) can lead to a missed identification; and in some instances, autolysis, the spontaneous disintegration of tissues after death, can occur.”

     The most chilling conclusion of their study, was one that should have been another dramatic beginning of the end of the HIV theory of AIDS: “It is possible that, as we suspect, all of the patients in this series had Kaposi’s sarcoma, although we could recognize it in only 94 percent. . . . The remarkable occurrence of Kaposi’s sarcoma in T-cell domains in virtually all of our cases suggests it may play a more important role in the pathogenesis of AIDS than is generally appreciated. We believe that Kaposi’s sarcoma contributes to the deterioration of cellular immunity seen in patients with AIDS by invasive destruction of T-cell domains, as in lymphoma or Hodgkin’s disease.”

     Katie Leishman also reported on the story, in the January 28 issue of the Los Angeles Times, and Ostrom covered her piece: “The CDC explains these results, Leishman states, by postulating that a second epidemic must have started at the same time and in the same populations as the ‘AIDS’ epidemic. According to the CDC, HIV is still responsible for ‘everything except Kaposi’s sarcoma.’ ”

     Leishman wrote, in the L.A. Times, “One is driven to wonder whether the researchers might have been correct on the point they have abandoned—that Kaposi’s sarcoma and all the other dismaying symptoms of AIDS do indeed have the same cause—but are wrong in the one that they still cling to: that HIV is the cause of AIDS. To question this has been denounced as heresy. But it was also once heresy to question HIV’s role in Kaposi’s sarcoma.”

     Even Randy Shilts, one of the fiercest acolytes of the HIV para-digm, was shaken by the findings. According to Leishman’s report he said, “It is the strangest twist in terms of medical news in the epidemic in years. It calls into question everything—the existing paradigm for the epidemic, the direction of research treatment modalities, and even the integrity of the blood supply.”

     What this major development made us wonder about, at the Native, was whether there was an epidemic of unrecognized Kaposi’s sarcoma spreading throughout America and the rest of the world, an epidemic of a form of KS that was perhaps slower and different in its manifestations, perhaps mostly internal and not obvious on the skin. Was variable, chronic KS the real AIDS epidemic? We immediately wondered if chronic fatigue syndrome patients (who officially were HIV-negative) had a form of KS in their internal organs that nobody would even think to look for. Or dare to. 

Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up.

"Persons infected with KSHV can asymptomatically shed the virus. It is advised to practice safe sex with infected individuals and curtail activities where saliva might be shared during sexual activity."

This is what oral Kaposi's Sarcoma looks like.


Compare it to these crimson crescent lesions in the mouths of Chronic Fatigue Syndrome patients.

"Burke A. Cunha, MD, discovered what he called crimson crescents in the mouths of 80% of his CFS patients. After the word got out, Cunha received calls from other parts of the country. Physicians began telling him that they also were finding the crimson crescents in their patients once they looked for them."

Chronic Fatigue Syndrome patients may have undiagnosed internal Kaposi's Sarcoma. Susan Levine found HHV-8, the Kaposi's Sarcoma virus, in half of CFS patients she looked at.

Prevalence in the Cerebrospinal Fluid of the Following Infectious Agents in a Cohort of 12 CFS Subjects

Susan Levine

Published online: 04 Dec 2011

Over the last decade a wide variety of infectious agents has been associated with the chronic fatigue syndrome (CFS) as potential etiologies for this disorder by researchers from all over the world. Many of these agents are neurotrophic and have been linked previously to other diseases involving the central nervous system (CNS). Human herpes virus-6 (HHV-6), especially the B variant, has been found in autopsy specimens of patients who suffered from multiple sclerosis. Because patients with CFS manifest a wide range of symptoms involving the CNS as shown by abnormalities on brain MRIs, SPECT scans of the brain and results of tilt table testing we sought to determine the prevalence of HHV-6, HHV-8, Epstein-Barr virus (EBV), cytomegalovirus (CMV), Mycoplasma species, Chlamydia species, and Coxsackie virus in the spinal fluid of a group of 12 patients with CFS. Although we intended to search mainly for evidence of actively replicating HHV-6, a virus that has been associated by several researchers with this disorder, we found evidence of HHV-8, Chlamydia species, CMV and Coxsackie virus in 6/12 samples. Attempts were made to correlate the clinical presentations of each of these patients, especially the neurological exams and results of objective testing of the CNS, with the particular infectious agent isolated. It was also surprising to obtain such a relatively high yield of infectious agents on cell free specimens of spinal fluid that had not been centrifuged. Future research in spinal fluid analysis, in addition to testing tissue samples by polymerase chain reaction (PCR) and other direct viral isolation techniques will be important in characterizing subpopulations of CFS patients, especially those with involvement of the CNS.

Everything you wanted to know about Kaposi’s Sarcoma in Chronic Fatigue Syndrome patients and the growing CFS epidemic of HHV-8, one of the two or three viruses that may be causing Kaposi’s Sarcoma.

Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up, a bestseller on Amazon.

     Neenyah Ostrom began one of my favorite series of articles in the same issue. Titled “The Color Purple,” Ostrom reported, “Burke Cunha, M.D. who is chief of infectious disease at Winthrop-University Hospital (Mineola, Long Island), has described what he calls ‘crimson crescents’ that appear in the throats of more than 80 percent of chronic fatigue syndrome (CFS) patients. Cunha describes the crescents not only as ‘crimson,’ but ‘purplish.’ The reddish-purplish regions found in CFS patients’ throats sounded quite similar to KS (Kaposi’s sarcoma) in the throat, commented an ‘AIDS’ doctor [who wished to remain anonymous] to whom they were described. Is it possible that the crimson crescents observed in the throats of CFS patients are actually a type of KS?”      Ostrom raised the possibility that the lesions in the throats of CFS patients connected them to the theory that Florida researchers held about KS being the unrecognized but unifying central pathological event AIDS. As I previously reported, the Florida team, headed by Dr. George Hensley, had turned the AIDS paradigm upside down, by finding KS in nearly 100% of AIDS patients, when they explored the internal organs closely during autopsies of AIDS patients. Their fascinating work suggested that KS preceded AIDS and caused more of the immune problem in AIDS than previously thought.

     Basically, Ostrom was asking if the KS-like lesions, in the tonsils of [CFS]patients, were an indication that some kind of unrecognized indolent KS was present internally, something that physicians would not even be thinking about because of the conceptual wall that socially hostile epidemiology had built between AIDS and chronic fatigue syndrome. And the CFS patients were not particularly interested in finding out if they shared KS with AIDS patients.

    Ostrom went even further, in the July 20 issue, and speculated that the dramatic digestive problems in chronic fatigue syndrome were actually the result of the unrecognized chronic or slowly progressive KS in the CFS patients’ digestive tracts. Ostrom noted that Dr. Carol Jessop, who was talking to a group of patients at a chronic fatigue syndrome conference, said, “Almost all patients would say to me, ‘I was totally well until I got this [chronic fatigue syndrome],’ and yet, when I took their past medical histories, I found it wasn’t quite true. Now these aren’t disastrous problems. In fact, if they had gone to their physicians for any of these problems such as irritable bowel, diarrhea and constipation, abdominal cramping, bloating, flatulence, chronic constipation, heartburn, etc., their physician would probably just say, ‘Oh, take this’ and that would be it. So we as physicians didn’t relate to our patients that this was a problem, so they considered themselves to be totally healthy. Yet, if you look at the numbers, 89 percent of the [chronic fatigue syndrome] patients had irritable bowel syndrome, diarrhea alternating with constipation, and abdominal cramping pain episodically. Another 80 percent complained of constant gas, bloating and flatulence. It’s amazing that we can all meet in this room together.”       Ostrom wondered if “Jessop may have uncovered a fallacy in the prevailing wisdom of chronic fatigue syndrome: that it begins as a respiratory, flu-like illness. Instead, as she points out, it may be a digestive tract disturbance. Jessop’s statistic—that more than 80 percent of CFS patients complain of irritable bowel syndrome, abdominal pain, gas, bloating, etc.—corresponds to the more than 80 percent of CFS patients who exhibit a red-to-purplish crescent-shaped lesion in their throats. (Helot, Paul, in the New York Times Long Island edition, January 14, 1992) . . . What if the digestive problems described by the CFS patients are actually caused by KS in the gastrointestinal tract? According to the AIDS Treatment News, ‘The most common HIV-related causes of gastric symptoms include KS, lymphoma, and CMV [cytomegalovirus].’ And while KS is unusual in the esophagus, it ‘may occasionally be found there.’ KS also can cause colitis and diarrhea . . . in people with AIDS.” Ostrom noted, “Gastrointestinal symptoms, it is realized in retrospect, were among the first signs of the ‘AIDS’ epidemic; and, it now seems, were also among the first symptoms seen in the CFS epidemic. That observation raises what should be a relatively simple question to answer: Are the gastrointestinal symptoms in both patient populations caused, in part, by undetected KS?”

Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up, a bestseller on Amazon.

Important information about the Kaposi’s Sarcoma problem in Chronic Fatigue Syndrome

Whatever happened to the concern about controlling the Kaposi's Sarcoma Virus? What about all the infected Chronic Fatigue Syndrome patients?

Is Kaposi's Sarcoma responsible for the digestive disorders in Chronic Fatigue Syndrome?

HHV-8 is a Kaposi's Sarcoma cancer virus in many AIDS and Chronic Fatigue Syndrome patients and is spread by kissing but the CDC couldn't care less.

Company founded by Robert Gallo suggests 65% of gay men are infected with Kaposi's Sarcoma virus.

Coagulation issues may link Chronic Fatigue Syndrome, Kaposi's Sarcoma, and AIDS

Should Chronic Fatigue Syndrome be added to the spectrum of Kaposi's Sarcoma-Associated Herpesvirus, or Human Herpesvirus 8, Diseases?

Why Susan Levine may have done the world's most important research on Chronic Fatigue Syndrome.

Does HHV-8 viral load raise questions about the legitimacy of HIV viral load?

Can Chronic Fatigue Syndrome patients with internal Kaposi's Sarcoma pass it on to their partners?

Can most of the symptoms of Chronic Fatigue Syndrome described by Paul Cheney be attributed to internal Kaposi's Sarcoma?

Is Chronic Fatigue Syndrome Associated Kaposi's Sarcoma  (CFSKS) a diagnosis all doctors should become aware of?

Stanford University and Open Medicine Foundation should have a conference on diagnosing Kaposi's Sarcoma in Chronic Fatigue Syndrome.

Why are doctors not looking for Kaposi's Sarcoma in Chronic Fatigue Syndrome patients?

If Chronic Fatigue Syndrome involves HHV-8 and Kaposi's Sarcoma, scientists will have to ask if it came from pigs.

Does the Red Blood Cell Deformability Issue Link Chronic Fatigue Syndrome to Kaposi's Sarcoma and AIDS?

Will the Montoya cytokine study show that Chronic Fatigue Syndrome is Kaposi's Sarcoma Inflammatory Syndrome?

Is Chronic Fatigue Syndrome a Kaposi's Sarcoma inflammatory cytokine syndrome?

How Kaposi's Sarcoma almost undermined the HIV theory of AIDS

How did 50% of Chronic Fatigue Syndrome patients become infected with a Kaposi’s Sarcoma cancer virus?

Has the moment finally come to address the issue of Kaposi's Sarcoma in Chronic Fatigue Syndrome?

Oral Kaposi's Sarcoma looks like the Crimson Crescents in Chronic Fatigue Syndrome patients.

Were oral crimson crescents the first obvious sign of Kaposi's Sarcoma in Chronic Fatigue Syndrome patients?

Did Paul Cheney ever consider the possibility that Chronic Fatigue Syndrome patients have internal Kaposi's Sarcoma?

Is the red blood cell deformability issue another clue that Chronic Fatigue Syndrome is also a Kaposi's Sarcoma Syndrome?

Why is nobody warned about exposure to HHV-8, the Kaposi's Sarcoma virus that even patients with Chronic Fatigue Syndrome are sometimes infected with?

Do petechiae in Chronic Fatigue Syndrome connect it to Kaposi's Sarcoma, HHV-8, and AIDS?

Whatever is causing Kaposi's Sarcoma may be the real cause of Chronic Fatigue Syndrome and AIDS.

A massive epidemic of Kaposi's Sarcoma may be coming.

When Kaposi's Sarcoma almost turned AIDS upside down.

Human herpesvirus 6 activates lytic cycle replication of Kaposi's sarcoma-associated herpesvirus.

All AIDS patients have some form of Kaposi's Sarcoma in this study. Is the same true for Chronic Fatigue Syndrome?

Crimson crescents may suggest that all Chronic Fatigue Syndrome patients have Kaposi's Sarcoma.

Do all Chronic Fatigue Syndrome patients have an indolent form of Kaposi's Sarcoma?

Are these marks on the skin a sign of Kaposi’s Sarcoma in Chronic Fatigue Syndrome?

On autopsy, do the inflamed ganglia of Chronic Fatigue Syndrome patients resemble Kaposi's Sarcoma?

What people don't know about Kaposi's Sarcoma in Chronic Fatigue Syndrome and AIDS.

Do all Chronic Fatigue Syndrome patients show internal Kaposi's Sarcoma upon autopsy?

Decades ago, a New York newspaper sounded the alarm about Kaposi’s Sarcoma in Chronic Fatigue Syndrome. The book about that newspaper is now a must-read bestseller on Amazon. Purchase a hardcover, paperback, or Kindle version here.

Three Big Books

Two books on amazon

Everyone needs to know what the CDC is hiding about CFS and HHV-6. NEW YORK NATIVE contains both volumes of THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP. The print version is $23. Only $7.98 in Kindle.

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