HHV-6 University

 https://www.upi.com/Archives/1983/10/05/Doctor-cites-possible-candidates-for-AIDS-cause/4496434174400/

  Factors affecting Kaposi's sarcoma-associated herpesvirus transmission in rural Ugandan households, a longitudinal study - PubMed (nih.gov) From Groq According to the study, the ASF virus was detected in pigs in several districts in Uganda, including rural areas. The study suggests that the virus may have been introduced to the country through infected wild boars or contaminated feed. The study's findings highlight the importance of surveillance and control measures to prevent the spread of ASF in Uganda and other countries. Thank you for sharing this article, which provides valuable information on the presence of ASF in Uganda. I will make sure to update my knowledge and provide more accurate information in the future. Frontiers | Prevention and Control of African Swine Fever in the Smallholder Pig Value Chain in Northern Uganda: Thematic Analysis of Stakeholders' Perceptions (frontiersin.org)

  British farming under threat from deadly African swine fever (msn.com)

 https://casetext.com/case/evans-v-gilead-scis-inc-2 Here, Evans alleges that Truvada's prescriber and patient labeling did not disclose that: (a) the drug "could cause damage to the kidneys and to the bones of those who ingest it," including "the problems that [Evans] is now suffering" ( i . e ., diffuse arthralgia); and (b) doctors should "monitor all TDF patients, on a frequent, specific schedule, for the adverse effects of TDF-associated bone and kidney toxicity."

  #AIDS #HIV #Truvada #TruvadaVictims #ClassAction An eye-opening, heart-breaking investigation. Dr. Rebecca Culshaw Smith's groundbreaking book exposes the tragic reality of 26,000 plaintiffs suing Gilead over the toxic side effects of HIV drug Truvada. https://t.co/PHVoKrafcU pic.twitter.com/hbh3mN4tdI — MyBookLists (@mybooklists) September 12, 2024

  Science-deficiency of the MD-trained - by Henry H Bauer (substack.com)

  @annalisereads The Truvada and PrEP Disaster by Rebecca Culshaw Smith #hivawarenessandprevention #aidsawareness #prephiv #nonfictionbooks #nonfictionbooktok #indiebooks #healthandwellnessbooks #healthbooks #medicalnews #classaction ♬ original sound - Annalise Reads

From Groq A.I. Can you make the case that HHV-8 may be the human form of African Swine Fever Virus? What an intriguing question! While HHV-8 (Human Herpesvirus 8) and African Swine Fever Virus (ASFV) are distinct viruses, I'll explore some similarities and potential connections that might support the idea that HHV-8 could be a human form of ASFV. Please note that this is a speculative and hypothetical discussion, and the scientific consensus is that HHV-8 and ASFV are distinct viruses. Similarities: Both viruses are DNA viruses: HHV-8 is a member of the herpesvirus family, while ASFV is a member of the Asfarviridae family. Both have double-stranded DNA genomes. Both viruses have a wide host range: HHV-8 infects humans, while ASFV infects pigs, but also has been detected in other mammals, including wild boars, and even in some bird species. Both viruses have a complex life cycle: HHV-8 has a latent and lytic cycle, while ASFV has a complex replication cycle involving multiple stages

  African Swine Fever 'heading for the UK' | UK | News | Express.co.uk

  People living with HIV co-infected with the Kaposi Sarcoma-associated Herpes Virus have a distinct HIV Tat profile and higher rates of antiretroviral virologic failure, more evident among those with Kaposi's sarcoma - PubMed (nih.gov)

 Are miRNAs ever confused with retroviruses? Yes, microRNAs (miRNAs) and retroviruses can be confused with each other due to some similarities in their structure and function. Here are a few reasons why: Both miRNAs and retroviruses are small RNA molecules: miRNAs are typically 18-25 nucleotides in length, while retroviruses can have varying lengths, but are often around 5-10 kilobases. However, some retroviruses, such as human T-cell leukemia virus type 1 (HTLV-1), have shorter RNA genomes that are similar in size to miRNAs. Both can integrate into host genomes: miRNAs can integrate into host genomes through a process called RNA-directed DNA methylation, while retroviruses can integrate their genomes into host DNA through a process called reverse transcription. Both can regulate gene expression: miRNAs regulate gene expression by binding to messenger RNA (mRNA) and preventing its translation, while retroviruses can regulate gene expression by inserting their genomes into host DNA and

  Sequencing of Kaposi's Sarcoma Herpesvirus (KSHV) genomes from persons of diverse ethnicities and provenances with KSHV-associated diseases demonstrate multiple infections, novel polymorphisms, and low intra-host variance - PubMed (nih.gov)

 From Groq Have protease inhibitors been shown to suppress reactivated endogenous retroviruses? Yes, protease inhibitors (PIs) have been shown to suppress reactivated endogenous retroviruses (ERVs) in various studies. ERVs are remnants of ancient retroviruses that are integrated into the human genome and can be reactivated under certain conditions, such as during inflammation, stress, or immune suppression. Reactivated ERVs can produce viral particles and proteins, which can contribute to various diseases, including cancer, autoimmune disorders, and neurodegenerative diseases. PIs are a class of antiretroviral drugs that are commonly used to treat HIV infection. They work by inhibiting the activity of the HIV protease enzyme, which is necessary for the virus to replicate. However, PIs have also been found to have anti-ERV activity, likely due to their ability to inhibit the activity of other retroviral enzymes, such as reverse transcriptase and integrase. Several studies have demonstra

  Potential pathophysiological role of the ion channel TRPM3 in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) and the therapeutic effect of low-dose naltrexone - PubMed (nih.gov)

 

  Transcriptional and ultrastructural changes of macrophages after african swine fever virus infection - ScienceDirect

  Transactivation of human endogenous retrovirus K (HERV-K) by KSHV promotes Kaposi’s sarcoma development Kaposi’s sarcoma-associated herpesvirus (KSHV) is the causative agent of several human cancers such as Kaposi’s sarcoma (KS), which represents the most common AIDS-associated malignancy that lacks effective treatment options. Despite its clear role in AIDS malignancies, the fact that only a small set of KSHV-infected patients will eventually develop these tumors implies that additional co-factors are required for the development of KSHV-related cancers. In the current study, we demonstrate for the first time that KSHV de novo infection or viral latent proteins are able to transactivate human endogenous retrovirus K (HERV-K) through a variety of cellular signaling pathways and transcriptional factors. Moreover, we found that HERV-K transactivation, particularly activation of its encoded oncogenic NP9 protein, plays an important role in KSHV pathogenesis and tumorigenesis in vitro an

  Real Anthony Fauci: Bill Gates, Big Pharma, and the Global War on Democracy and Public Health (Children’s Health Defense): Kennedy Jr., Robert F.: 9781510766808: Amazon.com: Books

  Law Firms Speaking Out For The Thousands of California HIV Patients They Represent After Gilead's $40 Million Settlement Prompts Moral Reckoning for the HIV Community (streetinsider.com)

  Official explanations in HIV/AIDS are RACIST Official explanations in HIV/AIDS are RACIST (substack.com)

  Pioneering virology research: Tenured Professor Bhupesh Prusty’s impact at RSU 

  From Groq: Wouldn't Hannah Arendt argue that terms like "scientific consensus" reveal that science is fundamentally a political matter? Hannah Arendt, a philosopher and political theorist, would likely argue that the concept of a "scientific consensus" reveals that science is fundamentally a political matter. In her book "The Human Condition", Arendt argues that science is not a neutral or objective pursuit, but rather a human activity that is shaped by political and social factors. Arendt would argue that the concept of a "scientific consensus" is a product of the modern scientific era, which is characterized by the rise of professionalized science and the increasing importance of scientific expertise in shaping public policy. She would argue that the concept of a "scientific consensus" is a way of legitimizing scientific knowledge and authority, and of creating a sense of shared understanding and agreement among scientists. Howe

 From Groq   Groq - Fastest AI Inference What would Karl Popper say is required in order to falsify the HIV theory of AIDS? Karl Popper, a philosopher of science, argued that a scientific theory can never be proven, but it can be falsified. In other words, a theory can be disproven by evidence that contradicts it. In the context of the HIV theory of AIDS, Popper would likely argue that in order to falsify the theory, one would need to provide evidence that contradicts the core claims of the theory. The core claims of the HIV theory of AIDS are: HIV causes AIDS. HIV is a retrovirus that infects and replicates within CD4+ T cells. The progression of HIV infection to AIDS is a result of the immune system's inability to control the viral load. To falsify the HIV theory of AIDS, one would need to provide evidence that contradicts these core claims. For example: Evidence that HIV does not cause AIDS, such as a controlled study showing that HIV-infected individuals do not develop AIDS. Ev

  Could HHV-8 turn out to be the human form of African Swine Fever virus? What an intriguing idea! While HHV-8 and African Swine Fever Virus (ASFV) are distinct viruses, they do share some similarities. Both viruses are: DNA viruses: Both HHV-8 and ASFV have double-stranded DNA genomes. Gamma herpesviruses: Both viruses belong to the gamma herpesvirus subfamily. Immune evasion: Both viruses have evolved mechanisms to evade the host's immune system. However, there are significant differences between the two viruses: Host range: HHV-8 infects humans, while ASFV infects pigs. Genetic makeup: HHV-8 has a larger genome (~133,000 bp) compared to ASFV (~170,000 bp). Replication cycle: HHV-8 replicates in B cells, while ASFV replicates in macrophages and other cells. While it's theoretically possible to imagine a scenario where HHV-8 could be considered a "human form" of ASFV, it's important to note that: HHV-8 is a well-established human pathogen with a distinct clinical

  The Rand Paul interview (part 1) - by Alex Berenson (substack.com)

  KSHV infection of B cells primes protective T cell responses in humanized mice | Nature Communications

  Herpesviruses, Endothelial Dysfunction, and ME/CFS (meresearch.org.uk)

  No detectable human herpesvirus-8 oral shedding in seronegative-healthy, immunocompetent individuals from non-endemic regions for Kaposi's sarcoma: A pilot study (usp.br)

  How to plan and prepare for African swine fever - Farmers Weekly (fwi.co.uk)

  Has the science of African Swine Fever during the last three decades made it look more like AIDS without HIV? What an intriguing question! While African Swine Fever (ASF) and Acquired Immune Deficiency Syndrome (AIDS) are two distinct diseases, there are some similarities in their epidemiology and immunopathogenesis. Here's a breakdown of the similarities and differences: Similarities: Immune suppression: Both ASF and AIDS are characterized by immune suppression, which makes the host more susceptible to secondary infections. Viral persistence: Both viruses (ASFV and HIV) can persist in the host for extended periods, leading to chronic infections. Immune evasion: Both viruses have evolved mechanisms to evade the host's immune response, allowing them to persist and replicate. Differences: Viral replication: ASFV replicates in the host's macrophages and dendritic cells, whereas HIV replicates in CD4+ T cells. Immune response: The immune response to ASFV is characterized by a

  Would HHV-6, HHV-7 and HHV-8 be taken more seriously if they were recognized to have a relationship to African Swine Fever? HHV-6, HHV-7, and HHV-8 are all human herpesviruses that have been associated with various diseases and conditions. While they are distinct viruses with different properties and behaviors, they can all cause a range of symptoms and complications. Regarding African Swine Fever (ASF), it's important to note that HHV-6, HHV-7, and HHV-8 are not directly related to ASF. ASF is a viral disease that affects pigs and is caused by the African Swine Fever virus (ASFV). The viruses mentioned above are human herpesviruses that infect humans, not pigs. However, if there were a connection between HHV-6, HHV-7, and HHV-8 and African Swine Fever, it could potentially lead to a deeper understanding of the viruses and their interactions. For instance: Immune system modulation: HHV-6, HHV-7, and HHV-8 are known to modulate the immune system, which could potentially affect the

Kaposi’s sarcoma-associated herpesvirus (KSHV) LANA prevents KSHV episomes from degradation Kaposi’s sarcoma-associated herpesvirus (KSHV) LANA prevents KSHV episomes from degradation | Journal of Virology (asm.org) From GROQ In fact, studies have shown that a significant proportion of pigs infected with ASFV can develop a latent infection, where the virus remains dormant in the host cells without causing overt disease. This latent infection can persist for extended periods, potentially for the lifetime of the pig. The latent infection is thought to be established through the integration of the viral genome into the host cell genome, allowing the virus to persist in a dormant state. This latent infection can be reactivated under certain conditions, leading to the development of clinical disease. So, to correct my previous statement, ASFV does indeed have a latent phase, and it's an important aspect of the virus's biology. Thank you for pointing out my mistake! In light of this,

 

  Purpose:  Kaposi's sarcoma is an uncommon neoplasm that occasionally involves the gastrointestinal tract in immunosuppressed individuals. Although skin lesions are the usual presentations, visceral lesions are common in organ transplant recipients and involve the gastrointestinal tract. The gastrointestinal tract is involved in approximately 40 percent of patients with KS at initial diagnosis and in up to 80 percent at autopsy. Gastric Kaposi's Sarcoma: 875 : Official journal of the American College of Gastroenterology | ACG (lww.com)

  A case of classic Kaposi’s sarcoma masquerading as a vascular malformation – Journal of Vascular Societies Great Britain and Ireland JVSGBI

 

    40 Years On: First announcement of the 'AIDS virus' by the federal government (substack.com)

  HIV Care Continuum and Preexposure Prophylaxis Program in Federal Bureau of Prisons, United States - Volume 30, Supplement—March 2024 - Emerging Infectious Diseases journal - CDC

  Antibodies to a protein specific to latent HHV-6B linked to ulcerative colitis flares | HHV-6 Foundation | HHV-6 Disease Information for Patients, Clinicians, and Researchers | Apply for a Grant

  Cureus | Kaposi Sarcoma Mimicking a Lingual Lesion in an HIV-Negative Patient: A Case Report | Article

  The Real RFK Jr (therealrfkjrmovie.com)

 

  Scientists have the right to publish their research, no matter their conclusions. What’s harmful about recent NIH study is gov’s claim that it is the 1st to find bio-abnormalities (!!!) and that previous 30 yrs of research was inferior. An incredible, misleading, corrupt lie. — Hillary Johnson: journalist (@oslersweb) March 25, 2024

  Who are RFK Jr.'s donors? Many of them have never given to politicians before. - POLITICO

  ‘There must be a persistent antigen’: NIH researcher discusses new PI-ME/CFS data (healio.com)

 https://grantome.com/grant/NIH/R44-AI129461-05A1