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Friday, September 30, 2016

Why HHV-6 should also be called "The Autism Virus"

Why HHV-6 should also be called
 "The Autism Virus"

1) HHV-6 is capable of causing most of the multi-systemic damage seen in Autism.

2) The HHV-6 epidemic and the rise in autism overlap.

3) Children with autism seem to improve when given HHV-6 antivirals.

4) HHV-6 explains why the CDC cannot tell the truth about autism. HHV-6 is involved with many more issues than autism. See the rest of the illnesses at HHV-6 University (hhv6.blogspot.com)


Thursday, September 15, 2016

A Special Gift for Visitors to HHV-6 University



A special song for anyone who has had to deal with bullies in science and medicine. Click below 
to listen on Spotify. 

Wednesday, September 14, 2016

Is a subclinical form of African Swine Fever Virus circulating in American pigs?

http://www.pigprogress.net/Finishers/Articles/2016/9/No-outbreaks-but-still-an-ASF-viral-presence-Why-2875555W/


Russian Scientist: ASF could become a human health risk


"The African swine fever (ASF) virus, may in the future become dangerous for humans, according to the head of the Russian Epidemiology Service, Chief State Sanitary Doctor Gennady Onishchenko, at the press-conference in St. Petersburg. According to him almost all viruses from time to time go through mutation processes which can give them some additional functions."

 http://www.pigprogress.net/Health-Diseases/Outbreaks/2013/7/ASF-could-become-a-human-health-risk-1308047W/

 

 



Background on African Swine Fever Virus as a human pathogen:

"African Swine fever is an endemic disease in sub-Saharan Africa and many other parts of the developing world. It is caused by the African Swine virus that primarily replicates in macrophages and monocytes leading to the impairment of the structure and function of the immune system of the infected organisms. Until now the African Swine epidemic continues to spread despite all efforts to contain it. Thus, there is an objective need for effective, safe and affordable preventive and therapeutic approaches, in particular for effective vaccines, to control and eventually eradicate this disease. Since the characteristic feature of the African Swine virus is to impair the immune system and to cause immune deficiencies in its hosts the development of vaccines and other therapeutic approaches against the African Swine virus has implications for other immune deficiencies or diseases. Several other viruses are also known to cause immunodeficiency-like syndromes in humans, including cytomegalovirus, Epstein Barr Virus and others. Moreover, a series of cases of so-called "idiopathic" immunodeficiencies have been documented that display CD4+T-lymphocytopenia with opportunistic infections, but show no evidence of HIV infection. Since antibodies for the African Swine virus have been detected in humans, the possibility of human infection with the African Swine virus exists and may thus far have escaped any systematic screening. Thus, any preventive and therapeutic approach to African Swine fever can have far-reaching implications to control immune deficiency conditions in humans."http://www.faqs.org/patents/app/20080207875

Detection of Novel Sequences Related to African Swine Fever Virus in Human Serum and Sewage.


Loh J, Zhao G, Presti RM, Holtz LR, Finkbeiner SR, Droit L, Villasana Z, Todd C, Pipas JM, Calgua B, Girones R, Wang D, Virgin HW.

Departments of Pathology & Immunology and Molecular Microbiology, Department of Medicine and Department of Pediatrics, Washington University School of Medicine, St. Louis, Missouri; Department of Biological Sciences, University of Pittsburgh, Pittsburgh, Pennsylvania; Department of Microbiology, Faculty of Biology, University of Barcelona, Barcelona, Spain.

"The family Asfarviridae contains only a single virus species, African swine fever virus (ASFV). ASFV is a viral agent with significant economic impact due to its devastating effects on populations of domesticated pigs during outbreaks, but has not been reported to infect humans. We report here the discovery of novel viral sequences in human serum and sewage which are clearly related to the Asfarvirus family, but highly divergent from ASFV. Detection of these sequences suggests that greater genetic diversity may exist among Asfarviruses than previously thought, and raises the possibility that human infection by Asfarviruses may occur."
http://www.ncbi.nlm.nih.gov/pubmed/19812170?dopt=Abstract

African Swine Fever Virus (Asfarviridae) sequences found in people with febrile illnesses

Abstract
Virus Identification in Unknown Tropical Febrile Illness Cases Using Deep Sequencing
Dengue virus is an emerging infectious agent that infects an estimated 50–100 million people annually worldwide, yet current diagnostic practices cannot detect an etiologic pathogen in ∼40% of dengue-like illnesses. Metagenomic approaches to pathogen detection, such as viral microarrays and deep sequencing, are promising tools to address emerging and non-diagnosable disease challenges. In this study, we used the Virochip microarray and deep sequencing to characterize the spectrum of viruses present in human sera from 123 Nicaraguan patients presenting with dengue-like symptoms but testing negative for dengue virus. We utilized a barcoding strategy to simultaneously deep sequence multiple serum specimens, generating on average over 1 million reads per sample. We then implemented a stepwise bioinformatic filtering pipeline to remove the majority of human and low-quality sequences to improve the speed and accuracy of subsequent unbiased database searches. By deep sequencing, we were able to detect virus sequence in 37% (45/123) of previously negative cases. These included 13 cases with Human Herpesvirus 6 sequences. Other samples contained sequences with similarity to sequences from viruses in the Herpesviridae, Flaviviridae, Circoviridae, Anelloviridae, Asfarviridae, and Parvoviridae families. In some cases, the putative viral sequences were virtually identical to known viruses, and in others they diverged, suggesting that they may derive from novel viruses. These results demonstrate the utility of unbiased metagenomic approaches in the detection of known and divergent viruses in the study of tropical febrile illness.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3274504/

Detection of African swine fever virus-like sequences in ponds in the Mississippi Delta through metagenomic sequencing

" . .. further study is needed to characterize their potential risks to both public health and agricultural development."

http://link.springer.com/article/10.1007%2Fs11262-013-0878-2

ASF virus, adapted to grow in VERO cells, produces a strong cytopathic effect in human macrophages leading to cell destruction.

http://vir.sgmjournals.org/content/34/3/455.short


Monday, September 12, 2016

The Davis/Naviaux Cover-up of the HHV-6 Epidemic

Two new scientists are now doing Anthony Fauci's work for him. They will have to work overtime to keep the HHV-6 epidemic hidden behind their scientific sounding  B.S. While Rome burns Davis and Naviaux fiddle.
 http://www.openmedicinefoundation.org/2016/09/09/viruses-and-cfs-statement-by-ron-davis-and-bob-naviaux/

Sunday, September 11, 2016

Saturday, September 03, 2016

Will Emetine be an effective treatment for HHV-6, Chronic Fatigue Syndrome, and AIDS?

http://hhv-6foundation.org/antiviral-treatments/low-dose-emetine-shows-promise-as-an-antiviral

The Very First Post on this HHV-6 Website in 2005

Online Interviewer: Can the CDC and NIH be held
criminally responsible for their alleged criminal
acts?
Regush: Not likely, but it wouldn't hurt them to know
that some people feel that they're really very
negligent the way they've addressed CFS and FM.
I'd like to see some heads roll at some point,
particularly those in the position of power to fund,
for they've neglected the concerns of many millions of
Americans. Anyone who claims that any of these
illnesses that we're talking about are in someone's
head should resign immediately. There is no scientific
argument for that position, none whatsoever. There is
not even any way to prove that point scientifically
unless one knows what the mind is or how the brain
works.
This all in the head business is one of the most
disgraceful positions held by medical experts who draw
their salaries from the government. I would debate any
of them, anywhere, at the drop of a dime on this point
alone. Having spent many years researching brain
science and writing a book on the brain, I can't
believe it when I hear adult scientists dismiss
physical symptoms as being somewhere in the mind. I
think here there may actually be grounds in the future
for malpractice.
http://prohealth.com/library/showarticle.cfm?ID=2953&T=BOTH

Note: For some reason the above link was changed since 2005. But in 2005 it worked.

The Coming Paradigm Shift in HHV-6 and Chronic Fatigue Syndrome

"Some studies have shown Human Herpes 6 and Epstein-Barr virus can impair the function of the human immune system. If one or both of these viruses indeed cause ME/CFS, and they invade the entire body, including the gut, then they could also disrupt the immune function of the gut. So, conceivably, the viral infection created a favorable environment for the pro-inflammatory bacteria. As a result, all of the disruption in the microbiome could stem from the underlying viral infection.
In conclusion, we appear to be entering the type of shake-up in the field that Thomas Kuhn referred to as a “paradigm shift.” All of the elements are there. A well-established concept about a phenomenon fails to account for a growing number of non-conforming bits of evidence. The examination of the bits of evidence reveals a new theoretical model that better explains the phenomenon."
http://www.psychiatryadvisor.com/opinion/chronic-fatigue-syndrome-is-not-merely-dysbiosis/article/520401/

Breaking HHV-6 News: "Anything that takes the public eye or the medical community's effort away from treating ME/CFS as a viral illness is a disservice."

"Reduced diversity and altered composition of the gut microbiome in individuals with myalgic encephalomyelitis/chronic fatigue syndrome, seems to ignore the extensive research demonstrating a viral etiology for ME/CFS."

http://www.psychiatryadvisor.com/opinion/chronic-fatigue-syndrome-is-not-merely-dysbiosis/article/520401/

Wake up CDC and NIH!

It's time for the NIH and the CDC to stop giving HHV-6 the silent treatment.

It's Time to Change the Name of HHV-6!

Isn't it time to change the name of HHV-6 to Acquired Immunodeficiency Virus or AIV?

Words matter. What we call things can change the world. As this song points out:

 

Friday, September 02, 2016

"We will test the hypothesis that ME/CFS results from poorly controlled infection with one or more human herpesviruses"

https://projectreporter.nih.gov/project_info_description.cfm?aid=9182215

Will this new study show AIDS and Chronic Fatigue Syndrome are intertwined?

https://projectreporter.nih.gov/project_info_description.cfm?aid=9182215

Breaking News: "We further hypothesise that the causative viruses [of CFS] will be human herpesviruses"

"Our hypothesis is that Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) has a viral aetiology and that the characteristic persistent remitting and relapsing nature of the syndrome is the result of poorly controlled infection with one or more known viruses, possibly due to an underlying immune deficiency. We further hypothesise that the causative viruses will be human herpesviruses as they have the key characteristics of being highly prevalent, giving rise to lifelong infections, and undergoing prolonged periods of dormancy/latency with periodic reactivation at times of stress or immune compromise."
 https://projectreporter.nih.gov/project_info_description.cfm?aid=9182215


The end is coming!


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