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Monday, April 06, 2015

So far, in this article, I have listed a total of 20 factors that ASFV and HHV-6A have in common. Ten are common areas of symptomology and 10 are particular characteristics of the virus. What is just as significant is the absence of clinical evidence that shows a difference between the two viruses. The only real test to settle the issue is for researchers to attempt to infect a pig with blood from an AIDS patients with Lymphadenopathy and see if it brings on African Swine Fever. If the real virus that causes AIDS came from pigs, then infecting them with the AIDS virus (HHV-6A) should cause them to develop ASFV. If it does, it will be case closed. Are there any researchers reading this article that are willing to do an in-vivo test on live swine? America's Biggest Cover-up A book came out late in Nov., 1993, titled America's Biggest Cover-up, by Neenyah Ostrom. It alleges that HHV-6 (A) may be the primary causative factor in both AIDS and Chronic Fatigue Syndrome. There are two strains of HHV-6, variants A and B. HHV-6 (Variant B) is a common herpes virus and is not a life threatening infection. Ostrom, a prolific researcher and writer, has written articles for The New York Native on HHV-6 and other AIDS related topics for several years (1). Ostrom says the variant A strain of HHV-6 is the culprit in both CFS and in AIDS. Ostrom has interviewed most of the major researchers in the field, as well as countless patients and government officials. She has found many similarities between Chronic Fatigue Syndrome and AIDS and believes that they are part of the same epidemic. She argues that until their connection is admitted by top government researchers, there is little hope that real progress will be made in stopping these two expressions of the same syndrome. Ostrom also says in her book that HHV-6A is really the African Swine Fever virus (ASF). Ostrom reports that CFS patients have lost much of their B cell and natural killer (NK) cell protection and both groups are susceptible to night sweats and many forms of cancer, lymphomas and TB. The book is available at your local bookstore.

http://www.keephopealive.org/report10.html

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Both ASFV and HHV-6A are hemorrhagic (cause bleeding) (1) and the immune reaction to ASFV in pigs shows an absence of effective neutralizing antibodies. (2). Since Dr. Salvato found 98% of her patients with full blown AIDS had replication of HHV-6A in the cells, as determined by PCR. This is comparable evidence of an absence of effective neutralizing antibodies. Both ASFV and HHV-6 are Icosahedral viruses (3, 4) and both are envelope viruses (5, 6). Also, both viruses, ASFV and HHV-6A, mature in the cytoplasm (4, 5). The replication of ASFV and HHV-6 (A) is inhibited by Phosphonoacetic acid. (6, 7). The replication of both viruses is also inhibited by Phosphoformic acid (Foscarnet) (6, 8).

http://www.keephopealive.org/report10.html

Gallo, Carrigan, Knox and other researchers have identified HHV-6A as a DNA lymphotropic virus that can infect T cells, B cells and monocytes and is systemicly infective and is found in most organs of persons with full blown AIDS. The Merck Veterinary Manual says this about ASFV: “This DNA virus replicates primarily in cells of the monocyte-macrophage system and is found in nearly all fluids and tissues of acutely infected pigs.” The fact that both viruses are DNA types and have tropism for infecting immune cells and can cause systemic infections adds further proof that both viruses are one and the same.

http://www.keephopealive.org/report10.html

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AN AIDS PATIENT REPORTS A FLARE-UP IN HIS KS LESIONS AFTER EATING PORK In Dec., 1995, I had a discussion with a PWA from Rio Rancho, NM, who has Kaposi Sarcoma. At the time, he told me that on three occasions when he ate pork, he had a flare-up and growth in his KS lesions. He did not get this effect from beef, turkey or chicken. He said: “when I eat pork, the KS goes wild.” He was not aware that research by Gallo and others have found the presence of HHV-6A in KS lesions. Since the ASF virus is known to systemicly infect pigs, it would not be surprising that eating pork would provide chow for ASFV(HHV-6A) in humans and adds more evidence that ASFV and HHV-6A are one and the same virus. This virus likes pork.

http://www.keephopealive.org/report10.html

ASFV and HHV-6A ARE THE SAME SIZE - 200 nm Both have lipid membranes. S. Zaki Salahuddin, Robert Gallo et al state that HBLV (HHV-6A) has “a lipid membrane and a diameter of the enveloped particle of about 200 nm.” (1). The African Swine Fever Virus (ASFV) has been identified as having a “lipid membrane” and a “diameter of 200 nm” by J.L Carrasosa et al (2) and S.S. Breese (3).

http://www.keephopealive.org/report10.html

An article by A Canals et al, published in Vet Microbiol, Nov., 1992, found that in peripheral blood mononuclear cells (PBMC) from pigs infected with ASFV, there was “progressive increase of the CD8 subset when the cells were stimulated with infective (ASF) virus.” In AIDS patients, one of the first things noticed in blood tests was the CD4/CD8 inversions. In healthy humans, HIV-, CD4s are always higher than CD8s. In AIDS, CD8s increase and are almost always higher than the CD4s. In Chronic Fatigue Syndrome (CFS), Dr. Patricia Salvato found 78% of her CFS infected with HHV-6. Dr. Nancy Klimas found a 19% elevation in CD8 counts in her CFS patients (1). In CFS and AIDS, African Swine Fever virus (sic:HHV-6A) increases CD8 counts, the same as it does in swine.

http://www.keephopealive.org/report10.html

Merck’s Manual also says the ASF virus is killed at 56 degrees Centigrade. An article appearing in The Lancet, Jan 26, 1985, by Montagnier, Spire, Dormat and Cherman was titled “Inactivation of Lymphadenopathy-associated virus by heat” said that “Lymphadenopathy associated virus (in AIDS) is inactivated by heating at 56 degrees C for 30 minutes. Since other researchers found that the HIV virus is inactivated at 60 degrees C and not 56, and it is known that HHV-6A is active in the lymph nodes (Gallo, Carrigan, Knox), the virus inactivated by Montagnier must not have been HIV, but rather HHV-6A.

http://www.keephopealive.org/report10.html

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What Merck’s is saying is plain English is that ASFV symptoms include fevers, low white blood cells, fluids in the lungs (pneumonia?), swollen lymph nodes, diarrhea, swollen veins that are purple (Kaposi’s sarcoma?), congested spleens and a kind of dementia (listless and uncoordinated). These symptoms are also found in AIDS in varying degrees. John Beldekas writing in The Lancet, March 8, 1986 stated: “Both AIDS and chronic ASFV are characterized by fever, hyperplastic lymph nodes, skin lesions, hypergammaglobulinaemia, immune-mediated pneumonia, thrombocytopenia, encephalopathy, high titre non-neutralizing antibody production, and lymphocyte depression.” Beldekas identified nine symptoms common to both viral infections.

http://www.keephopealive.org/report10.html

In August, 1986, John Beldekas was invited to go to the NCI and present his findings on the link between ASFV and AIDS, which he did. Beldekas gave samples of all his lab work to Gallo. Later, the government asked Beldekas to turn over all his reagents and lab work to the government, which he did. Beldekas had found ASFV presence in nine of 21 AIDS patients using two standard procedures. At the meeting, Gallo was reported saying: “we know it is not ASFV.” How could Gallo know this as he hadn’t done any of his own tests to look for ASFV? Two months later, Gallo published an article in Science (Oct 31, 1986) that he discovered a new possible co-factor in AIDS, a virus he called Human B Cell Lymphotropic Virus which he named HBLV. Like ASFV, HBLV infected B cells and also lived in macrophages. Did Gallo steal Beldekas’s ASF virus he found in AIDS patients and rename it HBLV? Later on, when Gallo found that HBLV could also infect other immune cells, he changed the name of HBLV to HHV-6. Eventually, Gallo identified his HBLV as the variant A strain of HHV-6 and called it a human herpesvirus.

http://www.keephopealive.org/report10.html

What may have been happening in the 12 AIDS patients who did not test positive for ASFV in the blood was that the virus may have been replicating in the cells.

http://www.keephopealive.org/report10.html

From 1983 to 1986, Jane Teas (Dept. of Pathology, Human Ecology Assn., Boston), John Beldekas (Boston University Medical School) and James R Hebert (Dept. of Epidemiology, Am Health Fdn, NY) searched for evidence of the ASF virus in AIDS patients. An article on their findings was published in The Lancet on March 8, 1986. What they found was the presence of ASFV in the blood of 9 of 21 AIDS patients using haemadsorption tests and found ASFV in 10 AIDS patients using immunofluorescence tests. Sixteen controls were used in the tests and one tested positive for ASFV. Beldekas also noted “giant cell formations, a characteristic of ASFV in swine cell cultures.” Beldekas indicated that possibly a new variant strain of ASFV was an infectious agent in AIDS and would cross react with Lisbon 60 strain which is known to cross react with all other strains of ASFV

http://www.keephopealive.org/report10.html

The first letter linking AIDS to African Swine Fever Virus (ASFV) was published in The Lancet, Apr. 23, 1983, by Jane Teas of the Harvard School of Public Health, Boston MA. Jane Teas and her colleague, John Beldekas, of the Boston University School of Medicine, found that the symptoms of ASFV in swine (pigs) was almost identical to symptomology found in AIDS patients. Common symptoms of both diseases are: fevers, swollen lymph nodes, pneumonia, skin lesions (like KS) and wasting syndrome. In pigs, ASFV is usually fatal, often within two weeks of the onset of symptoms, although there are some swine that survive ASFV infection.

http://www.keephopealive.org/report10.html

ASFV AND HHV-6A HAVE 20 FACTORS IN COMMON. AN IN-VIVO TEST IS NEEDED: INFECT SWINE WITH HHV-6A TO DETERMINE IF IT CAUSES AFRICAN SWINE FEVER

http://www.keephopealive.org/report10.html

The identification of an African swine fever gene with conserved helicase motifs and a striking homology to herpes virus origin binding protein, UL9.

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC309497/

Is HHV-6 Really African Swine Fever Virus in Drag?

http://jvi.asm.org/content/83/24/13019.full

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ProHealth's 2014 Advocate of the Year – Ryan Prior. Ryan Prior

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