Audible CFS book

An excerpt from THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP

CFS Book top ad

Kindle free trial

Monday, May 20, 2019

Cheney thought HHV-6 came from chickens. But it turned out to be pigs.



"Last winter, Cheney addressed a large CEBV support group in Incline Village. He was fresh from meetings with his collaborators at NIH. He brought with him startling news about chickens. “We think we know where HBLV may come from,” Cheney told the assembled CEBV sufferers and their families."

From "Journey Into Fear: Part Two The growing nightmare of Epstein-Barr Virus" by Hillary Johnson 

https://www.rollingstone.com/culture/culture-news/journey-into-fear-part-two-97373/

Porcine cytomegalovirus to be re-classified as a roseolovirus 


"Porcine CMV is an immunosuppressive virus that inhibits T-lymphocyte and macrophage immune functions, and like HHV-6A, it causes infertility. Porcine CMV infection also reduces the survival of pig xenotransplants. Porcine CMV (PCMV) will be designated as a roseolovirus by the International Committee on Taxonomy of Viruses in February.  The official name will be suid betaherpesvirus 2. A genomic study determined recently that PCMV is much closer to the roseoloviruses than to cytomegalovirus (Gu 2014). In fact, HHV-6 antibodies cross react with PCMV but HCMV antibodies do not."


https://hhv-6foundation.org/animal-models/porcine-cytomegalovirus-to-be-classified-as-a-roseolovirus


Read about the newspaper that warned that Chronic Fatigue Syndrome is connected to sick pigs.





Please support HHV-6 University by purchasing one or our books about the cover-up of the relationship between HHV-6 and Chronic Fatigue Syndrome, AIDS, and many other immunological illnesses. Click book to read a free excerpt.

Isn't it time to question the HIV theory of AIDS again?



Antiretroviral therapy (ART) is usually very effective at suppressing HIV in the body, allowing a person’s immune system to recover by preventing the virus from destroying CD4+ T cells (link is external). Scientists have now identified a rare, paradoxical response to ART known as extreme immune decline, or EXID. Five individuals evaluated at the National Institute of Allergy and Infectious Diseases (NIAID), part of the National Institutes of Health, experienced a significant decline in CD4+ T cell levels despite suppression of HIV below detectable levels for more than three years, according to a report published online today in JCI Insight.  The research team was led by Irini Sereti, M.D., chief of the HIV Pathogenesis Section in NIAID’s Laboratory of Immunoregulation, and Andrea Lisco, M.D., Ph.D.

The NIAID researchers found that the immune systems of people with EXID fared even worse than those of another subset of individuals defined as immunological-non-responders, or INRs, who respond inadequately to ART.  INR participants consistently taking ART for four years had CD4+ T cell counts that increased on average by 193 cells per microliter (µL) of blood.  Participants who responded normally to ART increased their CD4+ T cell count by more than twice that amount. In contrast, the five participants with EXID experienced an average decline of 157 CD4+ T cells/µL while consistently maintaining viral suppression on ART.

According to the NIAID team, there seems to be no single cause of EXID among the five individuals studied. Their analyses revealed that genes influencing immune cell activity and autoimmunity—the immune system attacking a body’s own healthy tissue—may play a role. Specifically, two of the individuals with EXID produced antibodies that attacked their own T cells, while two others had overactive cellular immune responses that lead to increased inflammation. All five participants with EXID had HIV strains other than clade B HIV (the most common strain circulating in North America and Europe), indicating that certain combinations of an individual’s genes and the HIV strain may be associated with EXID. While EXID is likely an extremely rare response to ART, the researchers indicate that studying this phenomenon may further illuminate CD4+ T cell reconstitution and inflammation in HIV disease and suggest possible treatment strategies for INRs and individuals with EXID.


https://www.nih.gov/news-events/news-releases/rare-cases-immune-system-fails-despite-hiv-suppression?utm_source=dlvr.it&utm_medium=twitter















Please support HHV-6 University by purchasing one or our books about the cover-up of the relationship between HHV-6 and Chronic Fatigue Syndrome, AIDS, and many other immunological illnesses. Click book to read a free excerpt.

The science of Chronic Fatigue Syndrome is as sick as the patients suffering from it.



The science of Chronic Fatigue Syndrome is suffering from a serious disease: AIDS fraud.


Only the truth can cure the disease that is infecting Chronic Fatigue Syndrome. These two books will get things moving in the right direction.






Please support HHV-6 University by purchasing one or our books about the cover-up of the relationship between HHV-6 and Chronic Fatigue Syndrome, AIDS, and many other immunological illnesses. Click book to read a free excerpt.

oct 19 ad

Individual chapters

Want to check out individual chapters of our best seller? Click books below for previews of each individual chapter of THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP available for only $2.99 on KINDLE or free on KINDLE Unlimited.

Popular Posts in the Last 30 Days

Blog Archive

Closing Argument Audible

African Swine Fever Novel Audible

Stonewall Audible