HHV-6 University

  High prevalence of antibodies to human herpesvirus 8 in relatives of patients with classic Kaposi's sarcoma from Sardinia A survey for antibodies to a recombinant small viral capsid antigen (sVCA) of human herpesvirus type 8 (HHV-8) was conducted in Sardinia, one of the world's highest incidence areas for classic Kaposi's sarcoma (KS). Prevalence of antibodies to HHV-8 sVCA was greatest in patients with KS (95%), followed by family members (39%) and a Sardinian control population age- and sex-matched to the relatives (11%). Within families, prevalence of antibodies was about equal among spouses, children, and siblings of KS patients, a finding that raises the possibilities of intrafamilial person-to-person or vertical transmission. Antibodies were detected 2-3 times more frequently in males than in females. The data show that prevalence of antibodies to HHV-8 sVCA correlates with the distribution of classic KS in a high- incidence area. Clustering of seroprevalence within

 In August, 1986, John Beldekas was invited to go to the NCI and present his findings on the link between ASFV [African Swine Fever virus] and AIDS, which he did. Beldekas gave samples of all his lab work to Gallo. Later, the government asked Beldekas to turn over all his reagents and lab work to the government, which he did. Beldekas had found ASFV presence in nine of 21 AIDS patients using two standard procedures. At the meeting, Gallo was reported saying: “we know it is not ASFV.” How could Gallo know this as he hadn’t done any of his own tests to look for ASFV? Two months later, Gallo published an article in Science (Oct 31, 1986) that he discovered a new possible co-factor in AIDS, a virus he called Human B Cell Lymphotropic Virus which he named HBLV. Like ASFV, HBLV infected B cells and also lived in macrophages. Did Gallo steal Beldekas’s ASF virus he found in AIDS patients and rename it HBLV? Later on, when Gallo found that HBLV could also infect other immune cells, he changed

  Dominican Republic restricts pig shipments, enlists military to fight African swine fever | Reuters In August, 1986, John Beldekas was invited to go to the NCI and present his findings on the link between ASFV [African Swine Fever virus] and AIDS, which he did. Beldekas gave samples of all his lab work to Gallo. Later, the government asked Beldekas to turn over all his reagents and lab work to the government, which he did. Beldekas had found ASFV presence in nine of 21 AIDS patients using two standard procedures. At the meeting, Gallo was reported saying: “we know it is not ASFV.” How could Gallo know this as he hadn’t done any of his own tests to look for ASFV? Two months later, Gallo published an article in Science (Oct 31, 1986) that he discovered a new possible co-factor in AIDS, a virus he called Human B Cell Lymphotropic Virus which he named HBLV. Like ASFV, HBLV infected B cells and also lived in macrophages. Did Gallo steal Beldekas’s ASF virus he found in AIDS patients and r

  The Seeker on Twitter: "Letter to Andersen Re: COVID Origin "We are very interested in understanding what happened on that call or what science came to light that caused such a dramatic change in your own hypothesis as to the engineering of COVID-19.” https://t.co/gZECZ5Qn1l https://t.co/g0yOtWxIcD" / Twitter

  Opinion | Covid Misinformation Comes From the Top, Too - The New York Times (nytimes.com)

  Rand Paul sends criminal referral to DOJ saying Fauci lied about gain-of-function research funding (msn.com)

  Germany Discovers First Cases of African Swine Fever in Farm Pigs | AgWeb J Virol .  2009 Dec;83(24):13019-25.   doi: 10.1128/JVI.00638-09.   Epub 2009 Oct 7. Detection of novel sequences related to african Swine Fever virus in human serum and sewage Joy Loh   1 ,  Guoyan Zhao ,  Rachel M Presti ,  Lori R Holtz ,  Stacy R Finkbeiner ,  Lindsay Droit ,  Zoilmar Villasana ,  Collin Todd ,  James M Pipas ,  Byron Calgua ,  Rosina Girones ,  David Wang ,  Herbert W Virgin Abstract The family Asfarviridae contains only a single virus species, African swine fever virus (ASFV). ASFV is a viral agent with significant economic impact due to its devastating effects on populations of domesticated pigs during outbreaks but has not been reported to infect humans. We report here the discovery of novel viral sequences in human serum and sewage which are clearly related to the asfarvirus family but highly divergent from ASFV. Detection of these sequences suggests that greater genetic diversity may e

  Thanks for the tweet. You can email me at sumathi.reddy@wsj.com. I saw the ME/CFS paper and would love to see/hear about your papers/research with long Covid patients. — Sumathi Reddy (@rddysum) July 14, 2021

  Are Latent Viruses Causing Long Covid-19 Symptoms? Patient Groups Push for Testing - WSJ

  Living with Chronic Fatigue Syndrome - Watch the full documentary | ARTE

  Are Autism Spectrum Disorders part of the Multisystemic HHV-6 Spectrum of Disorders? Is the Multisystemic HHV-6 Epidemic Causing the Dramatic Rise in Autism? Can the complications of Autism Spectrum Disorder be reduced by controlling Multisystemic HHV-6? Why won't the Centers for Disease Control tell the truth about the role of Multisystemic HHV-6 in Autism, Chronic Fatigue Syndrome, AIDS and many other serious neuroimmunological syndromes? What exactly is Multisystemic HHV-6? Is it really just a reactivated herpes virus or is it actually some other kind of lethal virus that can be passed from person to person and can become chromsomally integrated? Does it belong in a class by itself? Where did it come from? Is it a zoonose, a virus that is circulating in people and animals? "About six weeks back I was given the name of a woman who had approached Generation Rescue. She has a foundation which is working on the theory that the Human Herpes Virus #6 (HHV-6)

  The Enterovirus Theory of Disease Etiology in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Critical Review (nih.gov)

  The Enterovirus Theory of Disease Etiology in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Critical Review Adam J. O'Neal  and  Maureen R. Hanson * Abstract Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) is a complex, multi-system disease whose etiological basis has not been established. Enteroviruses (EVs) as a cause of ME/CFS have sometimes been proposed, as they are known agents of acute respiratory and gastrointestinal infections that may persist in secondary infection sites, including the central nervous system, muscle, and heart. To date, the body of research that has investigated enterovirus infections in relation to ME/CFS supports an increased prevalence of chronic or persistent enteroviral infections in ME/CFS patient cohorts than in healthy individuals. Nevertheless, inconsistent results have fueled a decline in related studies over the past two decades. This review covers the aspects of ME/CFS pathophysiology that are consistent with a chronic en