HHV-6 Transmission

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cartoon, hhv-6 , transmission, congress, fauci, public health,hiv, cfs, chronic fatigue syndrome
"Both intrauterine and sexual transmission of human herpesvirus (HHV)-6 and HHV-7 have been suggested."
http://www.ncbi.nlm.nih.gov/pubmed/17922393

"Transmission of this virus is believed to occur as a result of exposure to saliva."

http://www.emergingworlds.com/ch_viruses_detail.cfm?vPageid=41 

"Human herpesvirus 6 (HHV-6) is transmitted from parent to child in an integrated form and characterization of cases with chromosomally integrated HHV-6 DNA."

 http://www.ncbi.nlm.nih.gov/pubmed/15170644

"HHV-6 has a very high prevalence (close to 100% of the world’s population has been exposed). It is transmitted mainly by saliva. Transmission occurs usually within the first two years of life; primary infection is often associated with a febrile condition and sometimes with the onset of roseola (exanthem subitum). Two variants of the virus are known, HHV-6A and HHV-6B. HHV-6 is mainly lymphotropic, infecting a broad range of immune cells including T cells, monocytes, NK cells; however the virus can also infect many other tissues such as brain or liver."

http://www.redlabs.be/red-labs/our-science/viral-infections.php

"Virus replicated in the salivary glands and secreted in saliva is the epidemiologically proven source of transmission. Other routes of transmission have been suggested but remain to be proven."

http://wwwnc.cdc.gov/eid/article/5/3/99-0306_article 

"Infection with human herpes virus 6 (HHV-6) occurs in infancy or early childhood. Primary HHV-6 infection accounts for 10-20% of all febrile illnesses in infants and children less than 3 years of age seen in pediatric emergency departments. Serious complications have been described such as meningitis, encephalitis, hepatitis (including neonatal hepatitis), disseminated infection and hemophagocytic syndrome. At least one case of verified transmission from mother to foetus has been described. HHV-6 is closely related to CMV, which is transmitted from mother to infant in 0.5-2.0 of all pregnancies, most often as a result of reactivation of the virus."

http://www.nature.com/pr/journal/v41/n4s/full/pr19971488a.html

 "Respiratory transmission among persons with close contact with one another is the major route of primary infection."
 http://www.nature.com/jidsp/journal/v6/n3/full/5640056a.html


Unique route for human herpesvirus transmission: HHV-6 and chromosomal integration

 http://www.futuremedicine.com/doi/abs/10.2217/fvl.09.3


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http://www.journalofclinicalvirology.com/article/S1386-6532%2809%2900194-2/abstract Conclusion These results imply that HHV-6A, either in latent form or during acute infection, directly transactivates HERV-K18. This HERV-K18 induction may be mediated through IFN-α that is produced by the HHV-6A-infected cells. The functional implications of superantigen expression are discussed. Wikipedia on Superantigen: Superantigens (SAgs) are a class of antigens which cause non-specific activation of T-cells resulting in polyclonal T cell activation and massive cytokine release. SAgs can be produced by pathogenic microbes (including viruses , mycoplasma , and bacteria ) [1] as a defense mechanism against the immune system. [2] Compared to a normal antigen -induced T-cell response where .0001-.001% of the body’s T-cells are activated, these SAgs are capable of activating up to 25% of the body’s T-cells. [ citation needed ] Furthermore, Anti- CD3 and Anti- CD28 Antibodi...
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HHV-6 and Male infertility (from Perplexity)

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  Make the case that HHV-6 may be responsible for the fertility problems in men Make the case that HHV-6 may be responsible for the fertility problems in men There is a growing body of evidence suggesting that Human Herpesvirus 6 (HHV-6) may play a role in male fertility problems. Here is the case based on recent research findings: Prevalence in Semen and Sperm Binding:  Studies have identified HHV-6A/B in approximately 13.5% of semen samples from healthy sperm donors, a prevalence higher than that of most other herpesviruses in semen. Importantly, HHV-6B has been shown to specifically bind to the acrosome—the cap-like structure on the head of sperm. This means that the virus can directly interact with sperm cells and potentially travel with them into the female reproductive tract. Potential Mechanisms of Fertility Impairment:  The binding of HHV-6 to the sperm acrosome suggests the existence of a receptor for the virus on sperm, raising the possibility that this interact...
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