Pityriasis Rosea and HHV-6

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http://www.scribd.com/doc/256512858/Evaluation-of-Pityriasis-Rosea-Associated-With-5#scribd


Conclusion

This study provides further evidence for HHV-6 and HHV-7 as etiological factors inpityriasis rosea, and detecting HHV-7 DNA in biopsy specimens from skin lesions isrecommended.


Pityriasis rosea

From Wikipedia, the free encyclopedia

 http://en.wikipedia.org/wiki/Pityriasis_rosea


Pityriasis rosea (also known as pityriasis rosea Gibert[1]) is a skin rash. It is benign but may inflict substantial discomfort in certain cases.[2] Classically, it begins with a single "herald patch" lesion, followed in 1 or 2 weeks by a generalized body rash lasting up to 12 weeks, however usually around 6 - 8.[3][4][5]

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https://forums.phoenixrising.me/index.php?threads/human-endogenous-retroviruses-neuro-disorders-avindra-nath-publication.43314/ HHV-6A infection induces expression of HERV-K18-encoded superantigen Albert K. Taia, Janos Lukab, Dharam Ablashic, Brigitte T. Huber published online 08 June 2009. Abstract Background The human endogenous retrovirus K-18 (HERV-K18) encodes a superantigen that causes deregulation of the immune system. This provirus is transcriptionally silent, but can be induced by Epstein–Barr virus (EBV) infection and IFN-α treatment. Objectives Since the herpesvirus EBV induces HERV-K18 expression in human B cells, it was of interest to determine if other herpesviruses would have similar HERV-K18 transactivation properties. Human herpesvirus (HHV)-6A, a neurotropic virus associated with multiple sclerosis, was a logical candidate for these studies. Study design HSB2 cells (HHV-6-negative control), HSB2-ML cells (containing latent HHV-6A genome) and ...
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