Audible CFS book

An excerpt from THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP

CFS Book top ad

Kindle free trial

Wednesday, December 09, 2015

Is calling HHV-6 a"herpes virus" total bullshit that covers up its seriousness as the cause of a multisystemic epidemic?

The latent human herpesvirus-6A genome specifically integrates in telomeres of human chromosomes in vivo and in vitro

Abstract

Previous research has suggested that human herpesvirus-6 (HHV-6) may integrate into host cell chromosomes and be vertically transmitted in the germ line, but the evidence—primarily fluorescence in situ hybridization (FISH)—is indirect. We sought, first, to definitively test these two hypotheses. Peripheral blood mononuclear cells (PBMCs) were isolated from families in which several members, including at least one parent and child, had unusually high copy numbers of HHV-6 DNA per milliliter of blood. FISH confirmed that HHV-6 DNA colocalized with telomeric regions of one allele on chromosomes 17p13.3, 18q23, and 22q13.3, and that the integration site was identical among members of the same family. Integration of the HHV-6 genome into TTAGGG telomere repeats was confirmed by additional methods and sequencing of the integration site. Partial sequencing of the viral genome identified the same integrated HHV-6A strain within members of families, confirming vertical transmission of the viral genome. We next asked whether HHV-6A infection of naïve cell lines could lead to integration. Following infection of naïve Jjhan and HEK-293 cell lines by HHV-6, the virus integrated into telomeres. Reactivation of integrated HHV-6A virus from individuals’ PBMCs as well as cell lines was successfully accomplished by compounds known to induce latent herpesvirus replication. Finally, no circular episomal forms were detected even by PCR. Taken together, the data suggest that HHV-6 is unique among human herpesviruses: it specifically and efficiently integrates into telomeres of chromosomes during latency rather than forming episomes, and the integrated viral genome is capable of producing virions.
http://www.pnas.org/content/early/2010/03/01/0913586107.abstract

Authors

Jesse H. Arbucklea, Maria M. Medveczkya, Janos Lukab, Stephen H. Hadleya, Andrea Luegmayrc, Dharam Ablashid, Troy C. Lunde, Jakub Tolare, Kenny De Meirleirf, Jose G. Montoyag, Anthony L. Komaroffh, Peter F. Ambrosc, and Peter G. Medveczkya

Are HHV-6, HHV-7 and HHV-8 really Porcine Lymphotropic Herpes Viruses? And what is the real relationship between the so-called Porcine Lymphotropic Herpes Viruses (PLHV) and African Swine Fever Virus? Are they related?

Are pigs and pork infected with variants of HHV-6, HHV-7 and HHV-8? Is the pig version of these viruses causing the PRRS epidemic of immune deficiency in pigs all over the world? Is PRRS really a disease that is connected to African Swine Fever Virus? Are Porcine Lymhoroic Herps Viruses which are found in pigs afflicted with PRRS being confused with ASFV? Would pigs make the best model for HHV-6, HHV-7 and HHV-8 treatment experiments? A lot of issues need to be sorted out.

http://vir.sgmjournals.org/cgi/reprint/80/4/971.pdf
Robert Gallo's Laboratory's Research on the relationship between HHV-6 (originally called HBLV) and African Swine Fever Virus.

The story of John Beldekas, African Swine Fever Virus and HHV-6:

http://www.keephopealive.org/report10.html

Similarities Between Pig Virus and AIDS Explored in ABC News Commentary

Kaiser Health News


We are in the middle of the tragic HHV-6 epidemic. The big question about HHV-6 is this: What can't it do? What system in the body does it not infect and harm? In addition to AIDS it may be the key factor (or cause) in a number of current "mysterious" epidemics like chronic fatigue syndrome, autism and Morgellons by inducing expression of HERV-K18-encoded superantigen. Where did it come from? Who really discovered it? Robert Gallo or Jane Teas and John Beldekas? Technically speaking, is it really a herpes virus? Or is it actually a member of the asfaviridae family? Does it belong in a class by itself? Is it really a slow-acting zoonotic hemorrhagic virus? Is it also infecting animals? Are sick animals the original and current source of it? Which ones? What is the relationship of HHV-6, HHV-7 and HHV-8 to Porcine Lymphotropic Herpes Virus? Are pigs suffering from Porcine Reproductive and Respiratory Disease (PRRS) infected with some form of HHV-6, HHV-7 and HHV-8? Having established itself in complex diseases of the immune system in the late 20th Century, is HHV-6 becoming the biggest unrecognized human (and porcine) medical crisis of the 21st Century? Are we living in "The Age of HHV-6 Denialism" and when will it end? On this site you'll find lots of HHV-6 links and a great deal of food for thought.

We especially want to welcome scientists from Russia, China and other countries who are researching the relationship between African Swine Fever Virus and HHV-6, HHV-7 and HHV-8. Your work may help end this international tragedy


oct 19 ad

Individual chapters

Want to check out individual chapters of our best seller? Click books below for previews of each individual chapter of THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP available for only $2.99 on KINDLE or free on KINDLE Unlimited.

Popular Posts in the Last 30 Days

Blog Archive

Closing Argument Audible

African Swine Fever Novel Audible

Stonewall Audible