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Wednesday, October 23, 2019

Should American hotdogs be tested for African Swine Fever?


Hotdog samples, longaniza, tocino tested positive for ASF

https://news.mb.com.ph/2019/10/23/hotdog-samples-longaniza-tocino-tested-positive-for-asf-bai/



African Swine Fever is a big story already because, when and if it spreads to all of Western Europe, all of Asia and the USA (where it may already be in pigs), it will cause the collapse of a major portion of the agricultural export economies of the affected countries. We're talking about many billions of dollars of losses. And the problem is not temporary because those countries will be suspected of harboring the disease in their wild boar and ticks for decades to come. The disease could easily become endemic. 

But the issue is so much more important because of the disturbing body of evidence that shows that African Swine Fever Virus can infect humans (despite what authorities currently insist). Thus far, Europe and America's leading publications and journalists have failed to warn the public of the impending ASFV risk to their health. Here are the biggest African Swine Fever stories they have missed.

1. The African Swine Fever Vaccine for humans.

"African Swine fever is an endemic disease in sub-Saharan Africa and many other parts of the developing world. It is caused by the African Swine virus that primarily replicates in macrophages and monocytes leading to the impairment of the structure and function of the immune system of the infected organisms. Until now the African Swine epidemic continues to spread despite all efforts to contain it. Thus, there is an objective need for effective, safe and affordable preventive and therapeutic approaches, in particular for effective vaccines, to control and eventually eradicate this disease. Since the characteristic feature of the African Swine virus is to impair the immune system and to cause immune deficiencies in its hosts the development of vaccines and other therapeutic approaches against the African Swine virus has implications for other immune deficiencies or diseases. Several other viruses are also known to cause immunodeficiency-like syndromes in humans, including cytomegalovirus, Epstein Barr Virus and others. Moreover, a series of cases of so-called "idiopathic" immunodeficiencies have been documented that display CD4+T-lymphocytopenia with opportunistic infections, but show no evidence of HIV infection. Since antibodies for the African Swine virus have been detected in humans, the possibility of human infection with the African Swine virus exists and may thus far have escaped any systematic screening. Thus, any preventive and therapeutic approach to African Swine fever can have far-reaching implications to control immune deficiency conditions in humans."http://www.faqs.org/patents/app/20080207875

2. Evidence of African Swine Fever found in people with fevers.



Virus Identification in Unknown Tropical Febrile Illness Cases Using Deep Sequencing

Dengue virus is an emerging infectious agent that infects an estimated 50–100 million people annually worldwide, yet current diagnostic practices cannot detect an etiologic pathogen in ∼40% of dengue-like illnesses. Metagenomic approaches to pathogen detection, such as viral microarrays and deep sequencing, are promising tools to address emerging and non-diagnosable disease challenges. In this study, we used the Virochip microarray and deep sequencing to characterize the spectrum of viruses present in human sera from 123 Nicaraguan patients presenting with dengue-like symptoms but testing negative for dengue virus. We utilized a barcoding strategy to simultaneously deep sequence multiple serum specimens, generating on average over 1 million reads per sample. We then implemented a stepwise bioinformatic filtering pipeline to remove the majority of human and low-quality sequences to improve the speed and accuracy of subsequent unbiased database searches. By deep sequencing, we were able to detect virus sequence in 37% (45/123) of previously negative cases. These included 13 cases with Human Herpesvirus 6 sequences. Other samples contained sequences with similarity to sequences from viruses in the Herpesviridae, Flaviviridae, Circoviridae, Anelloviridae, Asfarviridae, and Parvoviridae families. In some cases, the putative viral sequences were virtually identical to known viruses, and in others they diverged, suggesting that they may derive from novel viruses. These results demonstrate the utility of unbiased metagenomic approaches in the detection of known and divergent viruses in the study of tropical febrile illness.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3274504/

3. A Russian Scientist warns that African Swine Fever could infect humans.

Russian Scientist: ASF could become a human health risk


"The African swine fever (ASF) virus, may in the future become dangerous for humans, according to the head of the Russian Epidemiology Service, Chief State Sanitary Doctor Gennady Onishchenko, at the press-conference in St. Petersburg. According to him almost all viruses from time to time go through mutation processes which can give them some additional functions."

 http://www.pigprogress.net/Health-Diseases/Outbreaks/2013/7/ASF-could-become-a-human-health-risk-1308047W/


4. Detection of Novel Sequences Related to African Swine Fever Virus in Human Serum and Sewage.
Loh J, Zhao G, Presti RM, Holtz LR, Finkbeiner SR, Droit L, Villasana Z, Todd C, Pipas JM, Calgua B, Girones R, Wang D, Virgin HW.

Departments of Pathology & Immunology and Molecular Microbiology, Department of Medicine and Department of Pediatrics, Washington University School of Medicine, St. Louis, Missouri; Department of Biological Sciences, University of Pittsburgh, Pittsburgh, Pennsylvania; Department of Microbiology, Faculty of Biology, University of Barcelona, Barcelona, Spain.

"The family Asfarviridae contains only a single virus species, African swine fever virus (ASFV). ASFV is a viral agent with significant economic impact due to its devastating effects on populations of domesticated pigs during outbreaks, but has not been reported to infect humans. We report here the discovery of novel viral sequences in human serum and sewage which are clearly related to the Asfarvirus family, but highly divergent from ASFV. Detection of these sequences suggests that greater genetic diversity may exist among Asfarviruses than previously thought, and raises the possibility that human infection by Asfarviruses may occur."
http://www.ncbi.nlm.nih.gov/pubmed/19812170?dopt=Abstract


5. How the American science Robert Gallo may have stolen the African Swine Fever research of a Boston University scientist and may have given African swine Fever the fraudulent new name of "HHV-6."

"In August, 1986, John Beldekas was invited to go to the NCI and present his findings on the link between ASFV [African Swine Fever virus] and AIDS, which he did. Beldekas gave samples of all his lab work to Gallo. Later, the government asked Beldekas to turn over all his reagents and lab work to the government, which he did. Beldekas had found ASFV presence in nine of 21 AIDS patients using two standard procedures. At the meeting, Gallo was reported saying: “we know it is not ASFV.” How could Gallo know this as he hadn’t done any of his own tests to look for ASFV?
Two months later, Gallo published an article in Science (Oct 31, 1986) that he discovered a new possible co-factor in AIDS, a virus he called Human B Cell Lymphotropic Virus which he named HBLV. Like ASFV, HBLV infected B cells and also lived in macrophages. Did Gallo steal Beldekas’s ASF virus he found in AIDS patients and rename it HBLV? Later on, when Gallo found that HBLV could also infect other immune cells, he changed the name of HBLV to HHV-6. Eventually, Gallo identified his HBLV as the variant A strain of HHV-6 and called it a human herpesvirus."
--Mark Konlee


http://www.keephopealive.org/report10.html

6. The epidemiology that suggests that African Swine Fever in people in Sardinia is misidentified as HHV-8.

The world's highest incidence of Kaposi's sarcoma occurs in Sardinia (Reference) Is it possible that it is due to the fact that African Swine Fever Virus is endemic on the island? (Reference) One study suggests that the incidence of K.S. in northern Sardinia is highest in a countryside area where people have contact with animals. (Reference) Given the high prevalence of HHV-8,--the so-called K.S. herpes virus--in Sardinia (Reference) is it at all possible that HHV-8 may have been misclassified and actually is a human-adapted form of African Swine Fever Virus? (ASFV has been at least visually mistaken for another herpes virus, CMV, in the past.)

A number of experiments could be conducted to explore this hypothesis. In addition to a direct comparison of ASFV and HHV-8, pigs with African Swine Fever Virus could be tested for sequences of HHV-8. People with Kaposi's sarcoma could be tested for sequences of African Swine Fever, including new Asfaviridae sequences recently discovered. (Reference) 


A comparison of the K.S. lesions in humans and ASFV lesions in pigs might be in order.Given that African Swine Fever is currently spreading in Russia and is now threatening Europe and China, (Reference) it would be useful to know whether people who are exposed to pigs with ASFV are at increased risk for HHV-8, Kaposi's sarcoma and the other pathologies associated with HHV-8. A study in sub-Saharan Africa where ASFV is endemic and HHV-8 is also endemic (Reference) might be useful. And areas of Russia where ASFV is spreading could be monitored closely for any signs of an increase of K.S. or HHV-8 infection and HHV-8 related pathologies.HHV-8 is an emerging health problem. HHV-8-associated K.S. is a significant problem in AIDS patients. It may also be the key to Chronic Fatigue Syndrome. HHV-8 has been found in the cerebrospinal fluid of 50% of Chronic Fatigue Syndrome patients. (Reference) HHV-8 has been linked to type 2 diabetes. (Reference) HHV-8 has been detected in B-cells in Castleman's disease and primary effusion lymphoma. (Reference).

If HHV-8 is a form of ASFV, it is possible that pigs might constitute a useful animal model for the study of possible treatments for K.S. and other pathologies associated with HHV-8. And if there is any relationship between ASFV and HHV-8, people may have to be warned to take special precautions around pigs in areas where there are ASFV outbreaks. And countries where undercooked pork is consumed (like Ukraine where salo is a staple) may need to alert the public to cook all pork products thoroughly during ASFV epidemics.

7. ASF virus, adapted to grow in VERO cells, produces a strong cytopathic effect in human macrophages leading to cell destruction.


8. A sick child tests positive for African Swine Fever virus.

9. Newspaper publisher writes The Chronic Fatigue Syndrome Epidemic Cover-up, a memoir about uncovering the African Swine Fever cover-up in America.

The Chronic Fatigue Syndrome Epidemic Cover-up details the investigative reporting of a New York Native that reveals the Centers for Disease Control and the United States Department of Agriculture lied about the presence of African Swine Fever in pigs and people.


10. Journalist pens The African Swine Fever Novel, an Orwellian novel warning about the consequences of an African Swine Fever Virus epidemic in humans.

The African Swine Fever Novel is available here

11. Prisoners fed meat infected with African Swine Fever






Is Kaposi's Sarcoma being transmitted to Chronic Fatigue Syndrome patients from their pets?

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4246215/









"Interestingly, recent observations show that feline immunodeficiency virus (FIV)–infected cats were developing cancers similar to those observed in humans with Epstein-Barr virus and Kaposi’s sarcoma­–associated herpesvirus. Subsequent polymerase chain reaction (PCR) screening identified 3 novel GHVs in domestic cats, bobcats, and pumas. PCR testing of nearly 1500 cats from the United States, Australia, Europe, Singapore, Japan, and Brazil has since estimated a 10% to 25% prevalence of the domestic cat GHV (known as Felis catus gammaherpesvirus 1 [FcaGHV1]). However, serologic studies suggest that infection rates are even higher than PCR results indicate."

https://www.americanveterinarian.com/news/new-feline-viruses-identified

Everything you wanted to know about Kaposi’s Sarcoma in Chronic Fatigue Syndrome patients and the growing CFS epidemic of HHV-8, one of the two or three viruses that may be causing Kaposi’s Sarcoma.







Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up, a bestseller on Amazon.



     Neenyah Ostrom began one of my favorite series of articles in the same issue. Titled “The Color Purple,” Ostrom reported, “Burke Cunha, M.D. who is chief of infectious disease at Winthrop-University Hospital (Mineola, Long Island), has described what he calls ‘crimson crescents’ that appear in the throats of more than 80 percent of chronic fatigue syndrome (CFS) patients. Cunha describes the crescents not only as ‘crimson,’ but ‘purplish.’ The reddish-purplish regions found in CFS patients’ throats sounded quite similar to KS (Kaposi’s sarcoma) in the throat, commented an ‘AIDS’ doctor [who wished to remain anonymous] to whom they were described. Is it possible that the crimson crescents observed in the throats of CFS patients are actually a type of KS?”      Ostrom raised the possibility that the lesions in the throats of CFS patients connected them to the theory that Florida researchers held about KS being the unrecognized but unifying central pathological event AIDS. As I previously reported, the Florida team, headed by Dr. George Hensley, had turned the AIDS paradigm upside down, by finding KS in nearly 100% of AIDS patients, when they explored the internal organs closely during autopsies of AIDS patients. Their fascinating work suggested that KS preceded AIDS and caused more of the immune problem in AIDS than previously thought.

     Basically, Ostrom was asking if the KS-like lesions, in the tonsils of [CFS]patients, were an indication that some kind of unrecognized indolent KS was present internally, something that physicians would not even be thinking about because of the conceptual wall that socially hostile epidemiology had built between AIDS and chronic fatigue syndrome. And the CFS patients were not particularly interested in finding out if they shared KS with AIDS patients.

    Ostrom went even further, in the July 20 issue, and speculated that the dramatic digestive problems in chronic fatigue syndrome were actually the result of the unrecognized chronic or slowly progressive KS in the CFS patients’ digestive tracts. Ostrom noted that Dr. Carol Jessop, who was talking to a group of patients at a chronic fatigue syndrome conference, said, “Almost all patients would say to me, ‘I was totally well until I got this [chronic fatigue syndrome],’ and yet, when I took their past medical histories, I found it wasn’t quite true. Now these aren’t disastrous problems. In fact, if they had gone to their physicians for any of these problems such as irritable bowel, diarrhea and constipation, abdominal cramping, bloating, flatulence, chronic constipation, heartburn, etc., their physician would probably just say, ‘Oh, take this’ and that would be it. So we as physicians didn’t relate to our patients that this was a problem, so they considered themselves to be totally healthy. Yet, if you look at the numbers, 89 percent of the [chronic fatigue syndrome] patients had irritable bowel syndrome, diarrhea alternating with constipation, and abdominal cramping pain episodically. Another 80 percent complained of constant gas, bloating and flatulence. It’s amazing that we can all meet in this room together.”       Ostrom wondered if “Jessop may have uncovered a fallacy in the prevailing wisdom of chronic fatigue syndrome: that it begins as a respiratory, flu-like illness. Instead, as she points out, it may be a digestive tract disturbance. Jessop’s statistic—that more than 80 percent of CFS patients complain of irritable bowel syndrome, abdominal pain, gas, bloating, etc.—corresponds to the more than 80 percent of CFS patients who exhibit a red-to-purplish crescent-shaped lesion in their throats. (Helot, Paul, in the New York Times Long Island edition, January 14, 1992) . . . What if the digestive problems described by the CFS patients are actually caused by KS in the gastrointestinal tract? According to the AIDS Treatment News, ‘The most common HIV-related causes of gastric symptoms include KS, lymphoma, and CMV [cytomegalovirus].’ And while KS is unusual in the esophagus, it ‘may occasionally be found there.’ KS also can cause colitis and diarrhea . . . in people with AIDS.” Ostrom noted, “Gastrointestinal symptoms, it is realized in retrospect, were among the first signs of the ‘AIDS’ epidemic; and, it now seems, were also among the first symptoms seen in the CFS epidemic. That observation raises what should be a relatively simple question to answer: Are the gastrointestinal symptoms in both patient populations caused, in part, by undetected KS?”





Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up, a bestseller on Amazon.







Important information about the Kaposi’s Sarcoma problem in Chronic Fatigue Syndrome

Whatever happened to the concern about controlling the Kaposi's Sarcoma Virus? What about all the infected Chronic Fatigue Syndrome patients?


Is Kaposi's Sarcoma responsible for the digestive disorders in Chronic Fatigue Syndrome?


HHV-8 is a Kaposi's Sarcoma cancer virus in many AIDS and Chronic Fatigue Syndrome patients and is spread by kissing but the CDC couldn't care less.


Company founded by Robert Gallo suggests 65% of gay men are infected with Kaposi's Sarcoma virus.


Coagulation issues may link Chronic Fatigue Syndrome, Kaposi's Sarcoma, and AIDS


Should Chronic Fatigue Syndrome be added to the spectrum of Kaposi's Sarcoma-Associated Herpesvirus, or Human Herpesvirus 8, Diseases?


Why Susan Levine may have done the world's most important research on Chronic Fatigue Syndrome.


Does HHV-8 viral load raise questions about the legitimacy of HIV viral load?


Can Chronic Fatigue Syndrome patients with internal Kaposi's Sarcoma pass it on to their partners?


Can most of the symptoms of Chronic Fatigue Syndrome described by Paul Cheney be attributed to internal Kaposi's Sarcoma?


Is Chronic Fatigue Syndrome Associated Kaposi's Sarcoma  (CFSKS) a diagnosis all doctors should become aware of?


Stanford University and Open Medicine Foundation should have a conference on diagnosing Kaposi's Sarcoma in Chronic Fatigue Syndrome.


Why are doctors not looking for Kaposi's Sarcoma in Chronic Fatigue Syndrome patients?


If Chronic Fatigue Syndrome involves HHV-8 and Kaposi's Sarcoma, scientists will have to ask if it came from pigs.


Does the Red Blood Cell Deformability Issue Link Chronic Fatigue Syndrome to Kaposi's Sarcoma and AIDS?


Will the Montoya cytokine study show that Chronic Fatigue Syndrome is Kaposi's Sarcoma Inflammatory Syndrome?


Is Chronic Fatigue Syndrome a Kaposi's Sarcoma inflammatory cytokine syndrome?


How Kaposi's Sarcoma almost undermined the HIV theory of AIDS


How did 50% of Chronic Fatigue Syndrome patients become infected with a Kaposi’s Sarcoma cancer virus?


Has the moment finally come to address the issue of Kaposi's Sarcoma in Chronic Fatigue Syndrome?


Oral Kaposi's Sarcoma looks like the Crimson Crescents in Chronic Fatigue Syndrome patients.


Were oral crimson crescents the first obvious sign of Kaposi's Sarcoma in Chronic Fatigue Syndrome patients?


Did Paul Cheney ever consider the possibility that Chronic Fatigue Syndrome patients have internal Kaposi's Sarcoma?


Is the red blood cell deformability issue another clue that Chronic Fatigue Syndrome is also a Kaposi's Sarcoma Syndrome?


Why is nobody warned about exposure to HHV-8, the Kaposi's Sarcoma virus that even patients with Chronic Fatigue Syndrome are sometimes infected with?


Do petechiae in Chronic Fatigue Syndrome connect it to Kaposi's Sarcoma, HHV-8, and AIDS?


Whatever is causing Kaposi's Sarcoma may be the real cause of Chronic Fatigue Syndrome and AIDS.


A massive epidemic of Kaposi's Sarcoma may be coming.


When Kaposi's Sarcoma almost turned AIDS upside down.


Human herpesvirus 6 activates lytic cycle replication of Kaposi's sarcoma-associated herpesvirus.


All AIDS patients have some form of Kaposi's Sarcoma in this study. Is the same true for Chronic Fatigue Syndrome?


Crimson crescents may suggest that all Chronic Fatigue Syndrome patients have Kaposi's Sarcoma.


Do all Chronic Fatigue Syndrome patients have an indolent form of Kaposi's Sarcoma?


Are these marks on the skin a sign of Kaposi’s Sarcoma in Chronic Fatigue Syndrome?


On autopsy, do the inflamed ganglia of Chronic Fatigue Syndrome patients resemble Kaposi's Sarcoma?


What people don't know about Kaposi's Sarcoma in Chronic Fatigue Syndrome and AIDS.


Do all Chronic Fatigue Syndrome patients show internal Kaposi's Sarcoma upon autopsy?




Decades ago, a New York newspaper sounded the alarm about Kaposi’s Sarcoma in Chronic Fatigue Syndrome. The book about that newspaper is now a must-read bestseller on Amazon. Purchase a hardcover, paperback, or Kindle version here.






Will pigs make the best model for studying HHV-8 and Kaposi's Sarcoma in Chronic Fatigue Syndrome and AIDS patients?


https://www.ncbi.nlm.nih.gov/pubmed/25092613

High prevalence of antibodies to human herpesvirus 8 in relatives of patients with classic Kaposi's sarcoma from Sardinia.


http://www.ncbi.nlm.nih.gov/pubmed/9607855


Infection with human herpesvirus type 8 and Kaposi's sarcoma in Sardinia.

 http://www.ncbi.nlm.nih.gov/pubmed/16501902

 

 Epidemiology of HHV8 in Sardinian emigrants

 http://www.dsnm.univr.it/?ent=progetto&id=565

 Both ASFV and HHV-8 Interfere with apoptosis.

Are ASFV-infected Pigs the viral source of HHV-8 related Kaposi's Sarcoma in Sardinia? Is a ASFV-related Kaposi's Sarcoma epidemic possible in Russia where ASFV is spreading?

 https://hhv6.jottit.com/35._pigs_and_kaposi%27s_sarcoma_in_sardinia

The world's highest incidence of Kaposi's sarcoma occurs in Sardinia (Reference) Is it possible that it is due to the fact that African Swine Fever Virus is endemic on the island? (Reference) One study suggests that the incidence of K.S. in northern Sardinia is highest in a countryside area where people have contact with animals. (Reference) Given the high prevalence of HHV-8,--the so-called K.S. herpes virus--in Sardinia (Reference) is it at all possible that HHV-8 may have been misclassified and actually is a human-adapted form of African Swine Fever Virus? (ASFV has been at least visually mistaken for another herpes virus, CMV, in the past.)

A number of experiments could be conducted to explore this hypothesis. In addition to a direct comparison of ASFV and HHV-8, pigs with African Swine Fever Virus could be tested for sequences of HHV-8. People with Kaposi's sarcoma could be tested for sequences of African Swine Fever, including new Asfaviridae sequences recently discovered. (Reference) 

A comparison of the K.S. lesions in humans and ASFV lesions in pigs might be in order.Given that African Swine Fever is currently spreading in Russia and is now threatening Europe and China, (Reference) it would be useful to know whether people who are exposed to pigs with ASFV are at increased risk for HHV-8, Kaposi's sarcoma and the other pathologies associated with HHV-8. A study in sub-Saharan Africa where ASFV is endemic and HHV-8 is also endemic (Reference) might be useful. And areas of Russia where ASFV is spreading could be monitored closely for any signs of an increase of K.S. or HHV-8 infection and HHV-8 related pathologies.HHV-8 is an emerging health problem. HHV-8-associated K.S. is a significant problem in AIDS patients. It may also be the key to Chronic Fatigue Syndrome. HHV-8 has been found in the cerebrospinal fluid of 50% of Chronic Fatigue Syndrome patients. (Reference) HHV-8 has been linked to type 2 diabetes. (Reference) HHV-8 has been detected in B-cells in Castleman's disease and primary effusion lymphoma. (Reference).

If HHV-8 is a form of ASFV, it is possible that pigs might constitute a useful animal model for the study of possible treatments for K.S. and other pathologies associated with HHV-8. And if there is any relationship between ASFV and HHV-8, people may have to be warned to take special precautions around pigs in areas where there are ASFV outbreaks. And countries where undercooked pork is consumed (like Ukraine where salo is a staple) may need to alert the public to cook all pork products thoroughly during ASFV epidemics.

 

A number of years ago, Neenyah Ostrom reported in the New
York Native on the lesions in CFS patients which seem
to resemble Kaposi's Sarcoma (KS). The current
thinking is that a virus called HHV-8 is the cause of
KS. (Although HHV-6 has recently also been implicated
once again.) If KS is a problem in CFS (and we
suspect it is) then one should be able to find HHV-8 and
HHV-6 in CFS patients. Apparently, in this small
study, one can. Below is a rather explosive abstract:
Prevalence in the cerebrospinal fluid of the following
infectious agents in a cohort of 12 CFS subjects:
human herpes virus-6 and 8; chlamydia species;
mycoplasma species; EBV; CMV; and Coxsackievirus.
Levine, S.
Journal of Chronic Fatigue Syndrome, 2001, 9, 1/2,
41-51.
Abstract:
Over the last decade a wide variety of infectious
agents have been associated with the CFS as potential
etiologies for this disorder. Many of these agents are
neurotrophic and have been linked previously to other
diseases involving the central nervous system (CNS).
Human herpes virus-6 (HHV-6), especially the B
variant, has been found in autopsy specimens of
patients who suffered from MS. Because patients with
CFS manifest a wide range of symptoms involving the
CNS as shown by abnormalities on brain MRIs, SPECT
scans of the brain and results of tilt table testing
we sought to determine the prevalence of HHV-6, HHV-8,
Epstein-Barr Virus (EBV), cytomegalovirus (CMV),
mycoplasma species, chlamydia species, and Coxsackie
virus in the spinal fluid of a group of 12 patients
with CFS (CDC criteria '94).
We found evidence of HHV-6, HHV-8, chlamydia species,
CMV and Coxsackie virus in 6/12 samples. Plasma tests
were negative. It was surprising to obtain such a
relatively high yield of infectious agents in cell
free specimens of spinal fluid that had not been
centrifuged. Future research in spinal fluid analysis,
in addition to testing tissue samples by polymerase
chain reaction (PCR) and other direct viral isolation
techniques will be important in characterizing
subpopulations of CFS patients, especially those with
involvement of the CNS.
The low rate of isolation of HHV-6 may be related to
the lack of gross neurological findings in the
patients at the time of testing.
An overview of KS:http://www.thebody.com/nmai/ks.html
Except for those who have made a lifelong commitment
to denial, finding the so-called "KS virus" (HHV-8)
and the "supporting KS virus" (HHV-6) in CFIDS patients
should help settle the question of the overlapping
nature of the AIDS and CFIDS epidemics.
Isn't it time to take a closer look at those crimson
crescents in the throats of CFIDS patients?
More info on KS here.

Given that HHV-8 is part of the HHV-6 and HHV-7 family, this may be very important:

"The 19R Protein of HHV-6 has significant amino acid sequence homology . . . to a protein encoded by African Swine Fever Virus."

--Glenda L. Lawrence, John Nicholas and Bart G. Barrell
Journal of General virology (1995), 76, 147-152

 Click here for more information on this issue.

  animal model, asfv, cdc, HHV-6, hhv-7, HHV-8, pigs, PLHV, porcine herpesvirus, swine

 

 


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