"Fauci" has been published on Apple in German

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 Fauci on Apple Books


Publisher Description

Dieses kleine Buch ist ein Kapitel aus „The Chronic Fatigue Syndrome Epidemic Cover-up Volume Two“ (Die Vertuschung der Epidemie des Chronischen Erschöpfungssyndroms – Band zwei).


In diesem brisanten kleinen Buch beschreibt der erste Verleger, der seine Zeitung der Berichterstattung über AIDS und das Chronische Erschöpfungssyndrom gewidmet hat, die Rolle von Anthony Fauci bei der Vertuschung der Wahrheit über den Zusammenhang der beiden Epidemien.


Angesichts der Tatsache, dass Anthony Fauci im Zentrum einer der größten medizinischen Vertuschungen der Geschichte steht, ist es schockierend, dass irgendjemand ihm während der COVID-19-Pandemie sein Vertrauen schenkt.


Während fehlgeleitete Medienvertreter wie Laurie Garrett und Rachel Maddow Fauci als „großen Amerikaner“ bezeichnet haben, wird Dr. Fauci bald seinen Platz in der Geschichte als Hauptverantwortlicher eines wissenschaftlichen Ponzi-Systems einnehmen, das die Welt in eine dystopische medizinische Dunkelheit aus Betrug, Täuschung und Vernachlässigung gestürzt hat.


Dieses Buch ist ein unverzichtbares Kapitel aus The Chronic Fatigue Syndrome Epidemic Cover-up Volume Two (Die Vertuschung der Epidemie des Chronischen Erschöpfungssyndroms – Teil Zwei) mit einem neuen Nachwort, das den großen Schaden untersucht, den Faucis Ponzi-System der Chronic Fatigue Syndrome-Community, den mit „HIV/AIDS“ stigmatisierten Menschen und allen, die an den Viren leiden, die Faucis Vertuschung vor der Welt verbirgt, zugefügt hat: die HHV-6/7/8-Virusfamilie. Die Liste der potenziellen Opfer von Faucis medizinischem Ponzi-System umfasst praktisch jeden. Sogar die Gesundheit von Millionen von Ärzten und Krankenschwestern ist in Gefahr geraten.

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5. Conclusions To the best of our knowledge this is the first study to report increased expression of HERV-K, HERV-H, HERV-W and INF-β and INF-γ levels correlations, in the immune system of FM patients. Although the levels of these molecules may serve as biomarkers of FM and/or ME/CFS and/or biosensors of TE activation, the RT-qPCR overall estimation approach used in this pilot study may turn unspecific, broadly associating TE activity with neurological and inflammatory processes. Therefore, future efforts evaluating activation of particular HERVs or TE chromosomal sites should more precisely define disease-specific mechanistic information. Importantly, the model proposed here linking disease-specific epigenome modifications, TE activation, inflammation and an increased risk of cancer in individuals with compromised RNase activity ( Figure 6 ) might be applicable not only to FM and ME/CFS patients but in any individuals with similar molecular disorders. https://www.mdpi.com/1...
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