Book previews

All of these books are available on Kindle and Kindle Unlimited. Hillary Johnson called THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP "fascinating and important." It has been on the Amazon bestseller lists since the day it was published.

Wednesday, February 04, 2015

Will somebody please sent this cartoon to Rachel Maddow?

cartoon, cfs, rachel maddow, anthony fauci, hhv-6


 http://www.nbcnews.com/id/56185745/ns/msnbc-rachel_maddow_show/t/rachel-maddow-show-thursday-october-nd/#.VN9GyS6b7b4
MADDOW: Dr. Anthony Fauci, director of the National Institute of Allergy
and Infectious Diseases at the National Institutes of Health.
Great American, sir. Thank you very much for being with us tonight. I
really appreciate it.
FAUCI: Good to be with you. Thank you, Rachel.
MADDOW: Thank you.
Not like I get to bestow the title great American, but Tony Fauci is really
a big deal. A big deal that he agreed to come on this show. I`ve
impressed myself by the fact that we had him.
All right. We`ve got lots more ahead tonight. Stay with us.

Early CFS Menopause: One of HHV-6's Shades of Contagious Non-HIV AIDS?


Study finds link between early menopause and CFS



http://www.eurekalert.org/pub_releases/2015-02/tnam-sfl020315.php


Is HHV-6 a Pig Virus?

OINK!

Click here for posts on HHV-6 as a pig virus

Does HHV-6 cause Contagious Non-HIV AIDS (a.k.a. porcine CFS) in pigs?

Flashback: A Summary of HHV-6's Destructivenss

From an article by Abdel-Haq NahedM, Asmar BasimI
in The Indian Journal of Pediatrics (2004,Volume 71,
Issue 1, Page 89-96)
HHV-6 preferentially infects CD4+ T-lymphocytes;[17]
but can also infect other cell lines of epithelial,
fibroblastic and neuronal origins with different
efficiency.[18],[19],[20] The surface marker CD46 acts
as part of a co-receptor for infection by both HHV-6A
and HHV-6B.[21] Although CD46 is expressed by most
nucleated human cells and may explain the wide
cellular tropism of HHV-6, the virus does not infect
all CD46-positive cells. Following infection, HHV-6
persists and establishes latency in different cells
and organs including monocytes/macrophages, salivary
glands, the brain and the kidneys. Because the main
target cells for infection are the CD4-positive
lymphocytes and monocytes, HHV-6 infection has
important effects on the immune system. These include
enhancement of natural killer cells, inhibition of
T-cell proliferation, induction of cytokine release
such as tumor necrosis factor-alpha, and interleukin
1-beta as well as modifying the expression of key cell
receptors such as CD3, CD4 and CXCR4.[17],[22],[23]

http://ijppediatricsindia.org/article.asp?issn=0019-5456;year=2004;volume=71;issue=1;spage=89;epage=96;aulast=Abdel-Haq

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