An excerpt from THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP

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Saturday, March 14, 2015

Time to be Proactive!

rachel maddow, tony fauci, hhv-6, cfs, aids, covr-up, aids

















 http://www.nbcnews.com/id/56185745/ns/msnbc-rachel_maddow_show/t/rachel-maddow-show-thursday-october-nd/#.VN9GyS6b7b4
MADDOW: Dr. Anthony Fauci, director of the National Institute of Allergy
and Infectious Diseases at the National Institutes of Health.
Great American, sir. Thank you very much for being with us tonight. I
really appreciate it.
FAUCI: Good to be with you. Thank you, Rachel.
MADDOW: Thank you.
Not like I get to bestow the title great American, but Tony Fauci is really
a big deal. A big deal that he agreed to come on this show. I`ve
impressed myself by the fact that we had him.
All right. We`ve got lots more ahead tonight. Stay with us.

A Population-Based Study of Primary Human Herpesvirus 6 Infection

http://www.nejm.org/doi/full/10.1056/NEJMoa042207

Breaking News!

Laboratory and Clinical Aspects of Human Herpesvirus 6 Infections.


Abstract

Human herpesvirus 6 (HHV-6) is a widespread betaherpesvirus which is genetically related to human cytomegalovirus (HCMV) and now encompasses two different species: HHV-6A and HHV-6B. HHV-6 exhibits a wide cell tropism in vivo and, like other herpesviruses, induces a lifelong latent infection in humans. As a noticeable difference with respect to other human herpesviruses, genomic HHV-6 DNA is covalently integrated into the subtelomeric region of cell chromosomes (ciHHV-6) in about 1% of the general population. Although it is infrequent, this may be a confounding factor for the diagnosis of active viral infection. The diagnosis of HHV-6 infection is performed by both serologic and direct methods. The most prominent technique is the quantification of viral DNA in blood, other body fluids, and organs by means of real-time PCR. Many active HHV-6 infections, corresponding to primary infections, reactivations, or exogenous reinfections, are asymptomatic. However, the virus may be the cause of serious diseases, particularly in immunocompromised individuals. As emblematic examples of HHV-6 pathogenicity, exanthema subitum, a benign disease of infancy, is associated with primary infection, whereas further virus reactivations can induce severe encephalitis cases, particularly in hematopoietic stem cell transplant recipients. Generally speaking, the formal demonstration of the causative role of HHV-6 in many acute and chronic human diseases is difficult due to the ubiquitous nature of the virus, chronicity of infection, existence of two distinct species, and limitations of current investigational tools. The antiviral compounds ganciclovir, foscarnet, and cidofovir are effective against active HHV-6 infections, but the indications for treatment, as well as the conditions of drug administration, are not formally approved to date. There are still numerous pending questions about HHV-6 which should stimulate future research works on the pathophysiology, diagnosis, and therapy of this remarkable human virus.


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