An excerpt from THE CHRONIC FATIGUE SYNDROME EPIDEMIC COVER-UP

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Saturday, February 27, 2016

Oh no!


Do Paralysis and Birth Defects suggest that Zika is really Classical Swine Fever Flavirus contracted from pigs in the ars of Brazil where both Zika and Classical Swine Fever are occurring?

Classical Swine Fever (CSF) - Hog Cholera (HC)

Classical swine fever (CSF), otherwise known as hog cholera (HC) or just swine fever, is a specific viral disease of pigs. It affects no other species. It is a notifiable disease in most countries of the world. Importance of CSF
CSF is one of the most economically-damaging pandemic viral diseases of pigs in the world. Many governments take it very seriously and adopt strict control policies, which include compulsory vaccination or slaughter and eradication policies.
In a susceptible (unvaccinated) herd almost all the pigs are affected. It causes generalised disease, including fever, malaise, lack of appetite, diarrhoea, paralysis, abortion, mummification and the birth of shaking piglets. Mortality is high.
Fortunately, there is only one serotype of the virus and attenuated vaccines are highly effective. Also, it does not spread on the wind or on insects or birds so standard precautions of farm biosecurity should keep it out. However it persists in uncooked and cured meat and these should not be fed to pigs. 

 http://www.thepigsite.com/pighealth/article/447/classical-swine-fever-csf-hog-cholera-hc/

Is Zika really Classical Swine Fever which is still a problem in pigs in Brazil?

https://www.gov.uk/guidance/classical-swine-fever

How to spot classical swine fever

The signs of classical swine fever are very similar to African swine fever.
The main clinical signs are:
  • fever
  • loss of appetite
  • lack of energy
  • sudden death with few signs beforehand
Other signs can include:
  • vomiting
  • diarrhoea
  • red or dark skin, particularly on the ears and snout
  • swollen red eyes
  • laboured breathing and coughing
  • abortions, still-births and weak litters
  • nervous signs, eg convulsions and tremors in newborn piglets
  • weakness
There are several different strains of classical swine fever.
Pigs infected with mild strains may not become ill or show clinical signs.
Severe strains of the disease are generally fatal.

Zika: Fluid can build up inside a fetus' body for several reasons, Zecavati said. "The fluid typically collects in the heart, lung, abdomen and skin tissue," she said. "The fluid overwhelms the body's ability to function, leading to failure or collapse of major organs, resulting in mortality as high as 50%

http://www.usatoday.com/story/news/2016/02/25/study-zika-linked-extensive-birth-defects-throughout-body/80850442/

Does the transmission of Classical Swine Fever Flavivirus teach us about the real transmission issues in Brazilian Zika disease?

Classical Swine Fever - APHIS - US Department of Agriculture

https://www.aphis.usda.gov/...
Animal and Plant Health Inspection Service







Classical Swine Fever - The Center for Food Security and ...
www.cfsph.iastate.edu/Factsheets/pdfs/classical_swine_fever.pdf

Is Zika really a pig disease?

Visit Zika Virus University.

Put two and two together: Are pigs in Caera, Brazil the real source of the Zika problem?

Zika is a problem in Caera, Brazil:
http://www.theguardian.com/global-development/2015/dec/04/brazil-zika-virus-pregnancy-microcephaly-mosquito-rare-birth-defect

Classical Swine Fever Virus (also a Flavivirus like Zika) is also a problem in Caera, Brazil. It causes birth defects in pigs and rashes. Connect the dots. Do the research 

[PDF]Classical Swine Fever in Brazil: An Update - David Publishing 

www.davidpublishing.com/davidpublishing/.../2012091700000690.pdf


If you're in Brazil, urge you scientists to consider the possibility that Zika is really another flavivirus, Classical Swine Fever virus.

Read this description of Classical Swine Fever Virus (which is infecting pigs in areas of Brazil with the so-called Zika problem)

Molecular Characterization of Classical swine fever virus Involved in the Outbreak in Mizoram

 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3550764/

 Does this sound like Zika?

 "Highest mortality in young animals was observed during the outbreaks. Percent mortality in pre-weaned and weaned piglets was 50% while 10% in pigs above 1 year of age. The disease showed a variable course and clinical symptoms. The affected animals exhibited clinical signs of inappetance, high fever and constipation followed by diarrhea and haemorrhagic patches in skin, characteristic signs of classical swine fever. There were rashes in the belly, medial aspects of thigh and on the base of ears. Necrotic lesions developed on skin in later stages. Affected pigs showed staggering gait. Pregnant sows either aborted or delivered stillborn fetuses. Agalactia was most common sequele of farrowed sows. On post-mortem examination, pathological changes recorded were button ulcer, extensive haemorrhage in intestine, mesenteric lymph nodes, pharyngeal lymph node, and in palatine tonsils. Kidneys and urinary bladder showed haemorrhagic spots."

 

http://www.huffingtonpost.com/entry/zika-virus-news_us_56b1180ee4b08069c7a53f25

 5 Questions for Brazilian scientists and doctors about the Zika-related epidemic

1. Are areas with major pig population places where Zika-related disease and deformities are concentrated?

2. What is the overall health of pigs in Brazil and is it in any way affecting human health?

3. Are Classical Swine Fever and Zika-related diseases and deformities at all related?

4. Did Brazil ever really get rid of African Swine Fever in the 80s or has a low grade form of it been simmering in its pigs for three decades and is it now the underlying problem of Zika-related disease and deformities in humans? 

5. Is Zika Virus just a biomarker or opportunistic infection? Is some other agent the fundamental cause?

Friday, February 26, 2016

Memo to Brazilian visitors to this site concerned about Zika

If you're in Brazil, urge you scientists to consider the possibility that Zika is really another flavivirus, Classical Swine Fever virus.

Read this description of Classical Swine Fever Virus (which is infecting pigs in areas of Brazil with the so-called Zika problem)

Molecular Characterization of Classical swine fever virus Involved in the Outbreak in Mizoram

 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3550764/

 Does this sound like Zika?

 "Highest mortality in young animals was observed during the outbreaks. Percent mortality in pre-weaned and weaned piglets was 50% while 10% in pigs above 1 year of age. The disease showed a variable course and clinical symptoms. The affected animals exhibited clinical signs of inappetance, high fever and constipation followed by diarrhea and haemorrhagic patches in skin, characteristic signs of classical swine fever. There were rashes in the belly, medial aspects of thigh and on the base of ears. Necrotic lesions developed on skin in later stages. Affected pigs showed staggering gait. Pregnant sows either aborted or delivered stillborn fetuses. Agalactia was most common sequele of farrowed sows. On post-mortem examination, pathological changes recorded were button ulcer, extensive haemorrhage in intestine, mesenteric lymph nodes, pharyngeal lymph node, and in palatine tonsils. Kidneys and urinary bladder showed haemorrhagic spots."

With Zika cases growing rapidly in US, will anyone look at pigs as a possible reservoir????

http://www.wsj.com/articles/reports-of-zika-cases-growing-quickly-in-u-s-1456521970

How HHV-8, a virus found in 50% of Chronic Fatigue Syndrome patients, may be causing cancer

http://www.nature.com/bcj/journal/v6/n2/full/bcj20166a.html


Prevalence in the cerebrospinal fluid of the following infectious agents in a cohort of 12 CFS subjects: human herpes virus-6 and 8; chlamydia species; mycoplasma species; EBV; CMV; and Coxsackievirus.

Levine, S. Journal of Chronic Fatigue Syndrome, 2001, 9, 1/2, 41-51.

Abstract: Over the last decade a wide variety of infectious agents have been associated with the CFS as potential etiologies for this disorder. Many of these agents are neurotrophic and have been linked previously to other diseases involving the central nervous system (CNS). Human herpes virus-6 (HHV-6), especially the B variant, has been found in autopsy specimens of patients who suffered from MS. Because patients with CFS manifest a wide range of symptoms involving the CNS as shown by abnormalities on brain MRIs, SPECT scans of the brain and results of tilt table testing we sought to determine the prevalence of HHV-6, HHV-8, Epstein-Barr Virus (EBV), cytomegalovirus (CMV), mycoplasma species, chlamydia species, and Coxsackie virus in the spinal fluid of a group of 12 patients with CFS (CDC criteria '94).
We found evidence of HHV-6, HHV-8, chlamydia species, CMV and Coxsackie virus in 6/12 samples. Plasma tests were negative. It was surprising to obtain such a relatively high yield of infectious agents in cell free specimens of spinal fluid that had not been centrifuged. Future research in spinal fluid analysis, in addition to testing tissue samples by polymerase chain reaction (PCR) and other direct viral isolation techniques will be important in characterizing subpopulations of CFS patients, especially those with involvement of the CNS.


Want to know more about HHV-8 and what animal might be its natural host? Read this:

High prevalence of antibodies to human herpesvirus 8 in relatives of patients with classic Kaposi's sarcoma from Sardinia.


http://www.ncbi.nlm.nih.gov/pubmed/9607855


Infection with human herpesvirus type 8 and Kaposi's sarcoma in Sardinia.

 http://www.ncbi.nlm.nih.gov/pubmed/16501902

 

 Epidemiology of HHV8 in Sardinian emigrants

 http://www.dsnm.univr.it/?ent=progetto&id=565

 Both ASFV and HHV-8 Interfere with apoptosis.

Are ASFV-infected Pigs the viral source of HHV-8 related Kaposi's Sarcoma in Sardinia? Is a ASFV-related Kaposi's Sarcoma epidemic possible in Russia where ASFV is spreading?

 https://hhv6.jottit.com/35._pigs_and_kaposi%27s_sarcoma_in_sardinia

The world's highest incidence of Kaposi's sarcoma occurs in Sardinia (Reference) Is it possible that it is due to the fact that African Swine Fever Virus is endemic on the island? (Reference) One study suggests that the incidence of K.S. in northern Sardinia is highest in a countryside area where people have contact with animals. (Reference) Given the high prevalence of HHV-8,--the so-called K.S. herpes virus--in Sardinia (Reference) is it at all possible that HHV-8 may have been misclassified and actually is a human-adapted form of African Swine Fever Virus? (ASFV has been at least visually mistaken for another herpes virus, CMV, in the past.)

A number of experiments could be conducted to explore this hypothesis. In addition to a direct comparison of ASFV and HHV-8, pigs with African Swine Fever Virus could be tested for sequences of HHV-8. People with Kaposi's sarcoma could be tested for sequences of African Swine Fever, including new Asfaviridae sequences recently discovered. (Reference) 

A comparison of the K.S. lesions in humans and ASFV lesions in pigs might be in order.Given that African Swine Fever is currently spreading in Russia and is now threatening Europe and China, (Reference) it would be useful to know whether people who are exposed to pigs with ASFV are at increased risk for HHV-8, Kaposi's sarcoma and the other pathologies associated with HHV-8. A study in sub-Saharan Africa where ASFV is endemic and HHV-8 is also endemic (Reference) might be useful. And areas of Russia where ASFV is spreading could be monitored closely for any signs of an increase of K.S. or HHV-8 infection and HHV-8 related pathologies.HHV-8 is an emerging health problem. HHV-8-associated K.S. is a significant problem in AIDS patients. It may also be the key to Chronic Fatigue Syndrome. HHV-8 has been found in the cerebrospinal fluid of 50% of Chronic Fatigue Syndrome patients. (Reference) HHV-8 has been linked to type 2 diabetes. (Reference) HHV-8 has been detected in B-cells in Castleman's disease and primary effusion lymphoma. (Reference).

If HHV-8 is a form of ASFV, it is possible that pigs might constitute a useful animal model for the study of possible treatments for K.S. and other pathologies associated with HHV-8 like Chronic Fatigue Syndrome. And if there is any relationship between ASFV and HHV-8, people may have to be warned to take special precautions around pigs in areas where there are ASFV outbreaks. And countries where undercooked pork is consumed (like Ukraine where salo is a staple) may need to alert the public to cook all pork products thoroughly during ASFV epidemics.


 

Is this a clue that Zika problems is caused by Classical Swine Fever Virus in Brazil's pigs?


Zika Virus Linked to Stillbirth

http://news.yahoo.com/zika-virus-linked-stillbirth-163950050.html


Brazil Develops Contingency Plan for Classical Swine Fever

http://www.thepigsite.com/swinenews/37230/brazil-develops-contingency-plan-for-classical-swine-fever/


 "The latest cases were reported in Brazil in August 2009, in Amapá, Pará and Rio Grande do Norte."

 http://www.thepigsite.com/swinenews/41309/parts-of-brazil-one-step-closer-to-classical-swine-feverfree-status/

Abortion in Pigs


"Many agents that cause reproductive failure in sows produce a broad spectrum of sequelae, including abortions and weak neonates, as well as stillbirth, mummification, embryonic death, and infertility. Mummification is seen more frequently in swine than in many other species because of the large litter size. If only a few fetuses die, abortion rarely occurs; instead, mummies are delivered at term, along with live piglets or stillbirths. . . The major infectious causes of reproductive failure in pigs include porcine reproductive and respiratory syndrome virus, porcine parvovirus, pseudorabies virus, Japanese B encephalitis virus, classical swine fever virus, Leptospira spp, and Brucella suis

 http://www.merckvetmanual.com/mvm/reproductive_system/abortion_in_large_animals/abortion_in_pigs.html  

Are Zika babies suffering from a congenital infection of a new or old strain of HHV-6.

Are pigs in the Zika areas of Brazil infected with a new or old strain of HHV-6?

Are scientists even aware that HHV-6 may also be a porcine infection? 

Is the Guillain-Barre problem also a hint that the underlying problem is HHV-6?

HHV-6 has been isolated from various tissues, cells, and fluid in association with the following conditions:
"Mechanical transmission by vehicles and equipment, as well as by personnel (notably veterinarians) travelling between pig farms, are also significant means of spread of CSF [Classical Swine Fever Virus, a flavivirus like Zika] within infected areas. The persistence of CSFV within herds for long periods has been observed. Infections of sows during pregnancy with low to moderately virulent strains of CSFV may lead to in utero infections of fetuses. These infections lead to litters born persistently infected with CSFV that are carriers of the virus and source for new infections. Persistently infected carrier pigs usually do not show clinical signs but constantly shed CSFV into the environment. Therefore, it is particularly important to consider CSFV infections while investigating herds presenting with unexplained reproductive failures that include clinical manifestations in piglets such as congenital tremor or congenital abnormalities."
 http://www.merckvetmanual.com/mvm/generalized_conditions/classical_swine_fever/overview_of_classical_swine_fever.html


Classical Swine Fever in Brazil: An Update - David Publishing

www.davidpublishing.com/davidpublishing/.../2012091700000690.pdf
by TRP Freitas - ‎Cited by 1 - ‎Related articles
Aug 20, 2012 - Key words: Classical swine fever, CSF-outbreaks, Brazil. 1. Introduction ... economic losses to the swine industries and pig farmers for beyond ...

Are pigs in Recife, Brazil the real source of the Zika-related problem in Brazil?

World Cup Brazil 2014: Part 1 – Sao Paulo-Recife-Natal for USA vs. Ghana and Japan vs. Greece


Luckily I negotiated a price beforehand instead of going by the meter, as there was massive traffic. Recife, Brazil’s 5th largest city, with a population of about 5m in the metro area, has an amazing coast line, but the interior of the city is very poor.
I immediately could see why Brazilians had been protesting the world cup. There’s no highway that goes across the city. The roads are pocked with pot holes that are big enough to eat your car. There’s a decent homeless population living in the streets. And you can tell people are struggling to get by.
Because traffic was so bad, the taxi driver asked me if we could take an alternate route. I agreed and we went through neighborhoods instead of the main road. I saw feral pigs eating trash, kids wearing shorts and nothing else, dilapidated houses and people with just their basic needs being met."

 http://www.nathanlustig.com/2014/07/11/world-cup-brazil-2014-part-1-sao-paulo-recife-natal-for-usa-vs-ghana-and-japan-vs-greece/

 If the World Health Organization says that the reservoir of Zika Virus is "unknown," shouldn't scientists be checking the pigs in Brazil for antibodies to it?

  http://www.who.int/mediacentre/factsheets/zika/en/

Zika virus is an emerging mosquito-borne virus that was first identified in Uganda in 1947 in rhesus monkeys through a monitoring network of sylvatic yellow fever. It was subsequently identified in humans in 1952 in Uganda and the United Republic of Tanzania. Outbreaks of Zika virus disease have been recorded in Africa, the Americas, Asia and the Pacific.
  • Genre: Flavivirus
  • Vector: Aedes mosquitoes (which usually bite during the morning and late afternoon/evening hours)
  • Reservoir: Unknown

Wednesday, February 24, 2016

Unwelcome Questions for Zika Researchers

Are Zika babies suffering from a congenital infection of a new or old strain of HHV-6.

Are pigs in the Zika areas of Brazil infected with a new or old strain of HHV-6?

Are scientists even aware that HHV-6 may also be a porcine infection? 

Is the Guillain-Barre problem also a hint that the underlying problem is HHV-6?

HHV-6 has been isolated from various tissues, cells, and fluid in association with the following conditions:

Chronic Fatigue Syndrome patients have a new quack to contend with.

http://www.meaction.net/2016/02/20/nih-lead-clinical-investigator-thinks-cfs-is-psychosomatic/

Tuesday, February 23, 2016

The big question for Atlanta:

The CDC says 1 in 2 gay black men will be diagnosed with HIV, but how many white heterosexual women will be diagnosed with what Judy Mikovits calls "Non-HIV AIDS"?
 https://www.yahoo.com/news/cdc-1-2-gay-black-men-us-diagnosed-175618678.html

Read what Judy Mikovits has to say about Chronic Fatigue Syndrome here.

Join an army of truthtellers!

Wear one of these and be a part of the army of truthtellers waking the world up to the facts about HHV-6.

Sunday, February 21, 2016

HHV-6 Disease is the new normal.

"Almost everyone worldwide is infected with HHV-6 by age 3."
 http://www.fredhutch.org/en/news/center-news/2015/11/good-news-life-sciences-hill-consortium-grants-anderson.html

Please send an email to Dr. Francis Collins, the Director of the National Institutes of Health.



Francis S. Collins
Director of the National Institutes of Health 
9000 Rockville Pike
Bethesda, Maryland 20892

Dear Dr. Collins:

It's time that the public knew about all the diseases HHV-6 is causing in our society. The days of using the fraudulent HIV paradigm of AIDS to cover up the HHV-6 pandemic must come to an end!

You shouldn't try to control panic about HHV-6 by lying to the public.

I urge you to support the goals of International HHV-6 Protests and Teach-ins that will be taking place at universities all over the world during the next several years.
Those goals include the support of freedom of thought, speech and dissent in science in general and in research of HHV-6-related diseases in particular. HHV-6-releated diseases include so-called "AIDS" and "Chronic Fatigue Syndrome," but are by no means limited to them. I also urge you to declare your support of the Harvard Declaration of the HHV-6 Rights of Man.



The Harvard Declaration of the HHV-6 Rights of Man

1. The right not to be lied to by Anthony Fauci about the role of HHV-6 in AIDS.
2. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Chronic Fatigue Syndrome.
3. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Autism.
4.The right not to be lied to by Anthony Fauci about the role of HHV-6 in Multiple Sclerosis.
5. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Brain Cancer.
6. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Heart Disease.
7. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Encephalitis.
8. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Cognitive Dysfunction.
9. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Drug Hypersensitivity Syndrome.
10. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Bone Marrow Suppression.
11. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Lymphadenopathy.
12. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Colitis.
13. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Endocrine Disorders.
14. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Liver Disease.
15. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Hodgkin's Lymphoma.
16. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Glioma.
17. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Cervical Cancer.
18. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Hypogammaglobulinemia.
19. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Optic Neuritis.
20. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Microangiopathy.
21. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Mononucleosis.
22. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Uveitis.
23. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Stevens-Johnson Syndrome.
24. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Rhomboencephalitis.
25. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Limbic Encephalitis.
26. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Encephalomyelitis
27. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Pneumonitis.
28. The right not to be lied to by Anthony Fauci about the role of HHV-6 in GVHD.
29. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Ideopathic Pneumonia.
30. The right not to be lied to about by Anthony Fauci the role of HHV-6 in Pediatric Adrenocortical Tumors
31. The right not to be lied to by Anthony Fauci about the role of HHV-6 in the reactivation of endogenous retroviruses.
32. The right not to be lied to by Anthony Fauci about the impact of HHV-6 on T-Cells.
33. The right not to be lied to by Anthony Fauci about the impact of HHV-6 on B-Cells
34. The right not to be lied to by Anthony Fauci about the impact of HHV-6 on Epithelial Cells.
35. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 on Natural Killer Cells.
36. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 on Dendritic Cells.
37. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 infection of the brain.
 38. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 infection of the liver.
39. The right not to be lied to by Anthony Fauci about the ability of HHV-6 to affect cytokine production.
40. The right not to be lied to by Anthony Fauci about the ability of HHV-6 to affect Aortic and Heart Microvascular Endothelial cells.
41. The right not to be lied to by Anthony Fauci about the role of an HHV-6 cover-up in a massive HIV Fraud Ponzi Scheme that in a number of ways resembles the Tuskegee Syphilis Experiment and Nazi medicine.

Sincerely,

Help end Fauci's Tuskegee Syphilis Experiment!










mainImg
Please send an email to Dr. Francis Collins, the Director of the National Institutes of Health.



Francis S. Collins
Director of the National Institutes of Health 
9000 Rockville Pike
Bethesda, Maryland 20892

Dear Dr. Collins:

It's time that the public knew about all the diseases HHV-6 is causing in our society. The days of using the fraudulent HIV paradigm of AIDS to cover up the HHV-6 pandemic must come to an end!

You shouldn't try to control panic about HHV-6 by lying to the public.

I urge you to support the goals of International HHV-6 Protests and Teach-ins that will be taking place at universities all over the world during the next several years.
Those goals include the support of freedom of thought, speech and dissent in science in general and in research of HHV-6-related diseases in particular. HHV-6-releated diseases include so-called "AIDS" and "Chronic Fatigue Syndrome," but are by no means limited to them. I also urge you to declare your support of the Harvard Declaration of the HHV-6 Rights of Man.



The Harvard Declaration of the HHV-6 Rights of Man

1. The right not to be lied to by Anthony Fauci about the role of HHV-6 in AIDS.
2. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Chronic Fatigue Syndrome.
3. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Autism.
4.The right not to be lied to by Anthony Fauci about the role of HHV-6 in Multiple Sclerosis.
5. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Brain Cancer.
6. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Heart Disease.
7. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Encephalitis.
8. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Cognitive Dysfunction.
9. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Drug Hypersensitivity Syndrome.
10. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Bone Marrow Suppression.
11. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Lymphadenopathy.
12. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Colitis.
13. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Endocrine Disorders.
14. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Liver Disease.
15. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Hodgkin's Lymphoma.
16. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Glioma.
17. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Cervical Cancer.
18. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Hypogammaglobulinemia.
19. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Optic Neuritis.
20. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Microangiopathy.
21. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Mononucleosis.
22. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Uveitis.
23. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Stevens-Johnson Syndrome.
24. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Rhomboencephalitis.
25. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Limbic Encephalitis.
26. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Encephalomyelitis
27. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Pneumonitis.
28. The right not to be lied to by Anthony Fauci about the role of HHV-6 in GVHD.
29. The right not to be lied to by Anthony Fauci about the role of HHV-6 in Ideopathic Pneumonia.
30. The right not to be lied to about by Anthony Fauci the role of HHV-6 in Pediatric Adrenocortical Tumors
31. The right not to be lied to by Anthony Fauci about the role of HHV-6 in the reactivation of endogenous retroviruses.
32. The right not to be lied to by Anthony Fauci about the impact of HHV-6 on T-Cells.
33. The right not to be lied to by Anthony Fauci about the impact of HHV-6 on B-Cells
34. The right not to be lied to by Anthony Fauci about the impact of HHV-6 on Epithelial Cells.
35. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 on Natural Killer Cells.
36. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 on Dendritic Cells.
37. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 infection of the brain.
 38. The right not to be lied to by Anthony Fauci about the the impact of HHV-6 infection of the liver.
39. The right not to belied to by Anthony Fauci about the ability of HHV-6 to affect cytokine production.
40. The right not to be lied to by Anthony Fauci about the ability of HHV-6 to affect Aortic and Heart Microvascular Endothelial cells.
41. The right not to be lied to by Anthony Fauci about the role of an HHV-6 cover-up in a massive HIV Fraud Ponzi Scheme that in a number of ways resembles the Tuskegee Syphilis Experiment and Nazi medicine.

Sincerely,

The Big Question


Friday, February 19, 2016

List of misconceptions about Chronic Fatigue Syndrome leaves out the one about it NOT being contagious and caused by a transmissible agent.

http://spoonseeker.com/2016/02/17/101-misconceptions-about-m-e/

February 19, 2016


Read these pages first!

Thursday, February 18, 2016

After covering up the CFS relationship to AIDS for over three decades.

The CDC calls Chronic Fatigue Syndrome "a devastating disorder."
 http://www.cdc.gov/cdcgrandrounds/archives/2016/february2016.htm

 How Dan Peterson's Friend Mary Guinan Helped the CDC Corrupt the Hard Drive of AIDS and Chronic Fatigue Syndrome Research

     Historians who want to trace the series of missteps that led to the HHV-6 pandemic and Holocaust II may benefit from taking a close look at a little known researcher at the CDC who played a curious role in both of the supposedly separate AIDS and chronic fatigue syndrome epidemics. Her surprising inability to see an obvious link between the two syndromes may be one of the important seeds of the whole HHV-6 disaster. She is mentioned in both the Shilts history of the early AIDS epidemic and Hillary Johnson’s journalistic account of the CDC’s bungling of the epidemic of facetiously-labeled chronic fatigue syndrome.
     According to Shilts, Mary Guinan worked for James Curran in the CDC’s venereal disease division. She was the person who sent James Curran the first ill-fated report on the first cases of what would eventually be called “AIDS” in “homosexuals.” With fellow VD chasers Harrold Jaffee and Curran, she shared the CDC AIDS Task Force’s  preoccupation with venereal diseases epidemiology. She helped impose the CDC heterosexist venereal groupthink on the emerging data of what would eventually be gayified epidemiologically into “Gay Related Immunodeficiency (GRID).”
     Ironically, considering what turned out to be the role of HHV-6 in AIDS, Shilts reported that in 1981, “on a hunch, Guinan called a drug company that manufactured medicine for severe herpes infections. They told her about a New York City doctor who had been seeing . . . dreadful herpes infections in gay men.” (ATBPO p.72) Shilts wrote that “Guinan was shaken by her investigation. She was accustomed to dealing with venereal diseases, ailments for which you receive an injection and are cured. This was different. She couldn’t get the idea out of her head: There’s something out there that’s killing people. That was when Mary Guinan hoped against hope that they would find something environmental to link these cases together. God help us, she thought, if there’s a new contagion spreading such death.” (ATBPO p.72) One way that God certainly wasn’t helping was by having a VD-obsessed doctor and her colleagues trying to comprehend a pandemic that wasn’t, strictly speaking, venereal.
     In Shilts’s account of Guinan, seeing the epidemic through gay-obsessed lenses was a given. He wrote about one of her days in 1981: “It had been another typical day of gay cancer studies for Mary Guinan. She had wakened at 6 a.m to breakfast with gay doctors and community leaders and asked again and again, ‘What’s new in the community?’ What new element might have sparked this catastrophe.” (ATBPO p.82) It was just gay, gay, gay—24/7—for the AIDS Task Force. They simply couldn’t wash the gay out of their hair. It was one of those times when every gay person should have checked to see whether they still had their wallets. Someone was about to sell them a gay epidemiological bridge.
     As Shilts sympathetically presents Guinan, he inadvertently nails the whole CDC psychological and sociological bias problem: “Guinan felt helpless and frightened. This was the meanest disease she had ever encountered. She strained to consider every possible nuance of these peoples’ lives.” (ATBPO p.83) What she really meant was gay nuances of gay lives. It is supremely ironic that Shilts wrote, “The CDC, she knew, needed to work every hypothesis imaginable into the case-control study.” (ATBPO p.83) Every hypothesis imaginable? Not by a long shot. How about the hypothesis that these cases were just extreme versions of the DuBois 1980 Atlanta cases that the CDC had been informed about? The un-gay cases.
     The process of identifying the emergence of the epidemic in nongay drug users, as described in Shilts’s book, makes it clear how gaycentric the thinking of the pioneers of the AIDS epidemiological paradigm was: “At the CDC there was a reluctance to believe that intravenous drug users might be wrapped into the epidemic, and the New York physicians also seemed obsessed with the gay angle, Guinan thought. ‘He’s said he’s not homosexual but he must be,’ doctors would confide in her.” (ATBPO p.83) Everybody was becoming an expert on gayness in those days. Given the reluctance to even see connections in those cases of nongay drug-using outcasts, it should come as no surprise when years later anyone saw the obvious connections between the epidemics of AIDS and chronic fatigue syndrome they were treated like they were strictly out to lunch. The AIDS paradigm was fatefully and messily intertwined with all the psychological baggage of sexual titillation and repulsion. If the CDC was unprepared psychologically to see drug users “wrapped into the epidemic,” how about all the good clean living white heterosexuals with the AIDS-like permutations of the immune system that characterize chronic fatigue syndrome?
     Guinan’s San Francisco trip with Harold Jaffe to interview AIDS patients and heterosexual controls also revealed the CDC mindset: “The CDC staffers could tell gay from straight controls by the way they reacted to the questions about every aspect of their intimate sexual lives. Heterosexuals seemed offended at queries about the preferred sexual techniques, while gay interviewees chatted endlessly about them.” (ATBPO p.96) Oh those gays! A herd of chatty Cathies if ever there was one. Given the bias-laden epidemiology that this chattiness was about to imprison the gay community in, one is tempted to say that loose gay lips sank a proverbial legion of gay ships. If one were watching this on a screen in a movie theater, one would want to scream out to the clueless gay interviewees for their own sake, “For Heaven’s sake, shut up!”
     Guinan was one of the CDC researchers credited by Shilts with recognizing that hemophiliacs and blood transfusion recipients might ultimately also become victims of “gay pneumonia.” She also was one of the first to worry about the AIDS infection possibilities of “semen depositors.” (ATBPO p.132) Guinan cast a wide net: “No sooner had she convinced the CDC that intravenous drug users were indeed a category of GRID cases separate from gay men, then her field of investigations discovered the first reported GRID cases among prisoners and prostitutes.” (ATBPO p.132) Unfortunately epidemiological net wasn’t wide enough to catch the 1980 DuBois Atlanta Cases of immune dysfunction. Also unfortunately for her, she helped create the very consequential epidemiological urban myth of Patient Zero. She was the first person to come in contact with Gaetan Dugas the so-called gay Typhoid Mary who the CDC would turn into the “Patient Zero” or more appropriately, “Scapegoat Zero,” of the epidemic depending on your point of view. He would become an icon for all the venereal gaycentric thinking down at the CDC.
     In one of those amazing moments in Holocaust II in which a scientist comes so face-to-face with the truth but fails to see what is right before their eyes, Shilts reports that when Guinan was studying drug users, “blood sampling of the intravenous drug users also revealed that, although many were infected with cytomegalovirus, the viral strains were all different. This was strong evidence that the herpes virus had not developed some new virulent strain. No single strain emerged, lending further weight to Don Francis’s hypothesis that a new virus, not CMV was a work.” (ATBPO p.133) The CDC, in retrospect, was most likely eyeballing strains of an undiscovered virus that would be called HBLV when Gallo’s scientists supposedly “discovered” it in 1986. It was subsequently named HHV-6. In retrospect it is pretty obvious that the CDC was looking at HHV-6 but thinking it was only CMV. (And those who wanted to see a retrovirus would have been especially predisposed not to see a new DNA virus like HBLV/HHV-6.)
     It is interesting and perhaps revealing that Guinan and her colleagues could deal with the fact that the disease or syndrome manifested itself differently in gay men and drug users—presumably for reasons that would ultimately be figured out. But God forbid that anyone would subsequently suggest that even though there were differences in the manifestations of chronic fatigue syndrome and AIDS that they were essentially manifestations of the same agent and the same pandemic. Distinctions were not turned into differences where drug users and gays were concerned, but where the gays with AIDS and the middle-class straights with chronic fatigue syndrome were concerned, every distinction,—even the teeny-tiniest or most irrelevant kind—was immediately considered a dramatic how-dare-you-compare-these-apples-and-oranges difference. Such bogus thinking would be at the heart of the “chronic fatigue syndrome is not AIDS” paradigm which would guide public health through the next three decades.
     For all her good work Guinan was eventually rewarded with the position of assistant CDC director. Unfortunately for all the victims of HHV-6, what she did do at the CDC didn’t have as much impact on the well being of the world as what she did not do. It was Guinan in 1985 who got a call from Dan Peterson, a former colleague and one of the two doctors who are credited with recognizing an outbreak of the absurdly named “chronic fatigue syndrome” in their Lake Tahoe practice. According to Hillary Johnson, “The two had become friends during a shared stint at the at the University of Utah hospital in Salt lake City in 1976.” (OW p.31) Also, according to Johnson, “Peterson had frequently sought her counsel on different infectious disease cases; he had also struck her as a gifted diagnostician.’ (OW. P.31)
     Johnson reported that “Guinan listened as her former colleague described his Tahoe patients, her curiosity aroused by the possibility that this ailment, which three recent medical papers had described, was occurring in epidemic form. Previously, researchers had described it as a sporadic illness. She remembered too, that Atlanta clinician Richard DuBois had made a presentation to agency staff on the malady early in 1983, even proposing that the new mono-like syndrome might be a second epidemic of immune dysfunction rising concurrently with AIDS.” (OW p.31)
     Did this lead Guinan serendipitously into a more complicated epidemiological vision of a variable epidemic that included both what was called “AIDS” and “chronic fatigue syndrome”? Not on your life. These first CFS patients were not gay and not drug users. They were from medical practices that could be described as being devoted to folks who ride in the middle and front part of society’s bus. Such stark social differences would make it of no consequence or interest that study after study would show one immunological and neurological similarity after another between AIDS and chronic fatigue syndrome. Guinan had helped build a paradigm that was so gay, gay, gay and so socially radioactive that the links between AIDS and CFS would be willfully ignored, buried alive by denial, and through a kind of determined public health radio silence, for all intents and purposes, be covered-up big time.
     Ignoring the obvious, Guinan sent the future “CFS” patients of America on one of the greatest medical wild goose chases in history. According to Johnson, she passed the Peterson cases on to Larry Shonberger, chief of the CDC’s epidemiology within the Division of Viral and Ricketsial Diseases. Not surprisingly, Johnson reports that “Schonberger and his staff of epidemiologists had a mandate to monitor and occasionally investigate outbreaks of viral diseases, with the exception of AIDS, which by 1985 had been awarded a separate division and staff and more than half of the federal agency’s entire annual research budget.” (OW p.32) And so, because of Guinan’s phone call and her very questionable judgment, CFS research headed down exactly the wrong road
     Had Guinan wisely directed the Lake Tahoe cases in the direction of the CDC’s AIDS division back in 1985, there was still a chance that the political and medical apartheid of the “chronic fatigue syndrome is not AIDS’ paradigm and Holocaust II might not have been able to fully materialize. But AIDS had been so gayified and turned into such a sexual bogeyman and scarlet letter syndrome, that Guinan and everyone else at the CDC couldn’t for the life of them admit that average (i.e. heterosexual) Americans were coming down with any similar or related form of acquired immunodeficiency. Instead those people were given the whitewash of a diagnosis of chronic fatigue syndrome. Those good country people, to borrow a term from Flannery O’Connor, couldn’t in a million years be suffering from something that had at one time or another been called Gay Cancer, Gay Pneumonia, and Gay Related Immunodeficiency. After all, they weren’t gay.

Zika: "Or it could be something else."

 ". . . the final baffling effect of Zika. Is it causing Guillain-Barre syndrome, an inflammation of the nerves that can cause sometime-serious paralysis after any number of infections?"

http://www.nbcnews.com/storyline/zika-virus-outbreak/mysteries-zika-virus-stump-even-experts-n520816


Or are we looking at the new face of the HHV-6 pandemic?

 Human Herpesvirus 6 and Neuroinflammation
 http://www.hindawi.com/journals/isrn/2013/834890/

More evidence Chronic Fatigue Syndrome and AIDS are part of the same epidemic?

A Preliminary Comparative Assessment of the Role of CD8+ T Cells in Chronic Fatigue Syndrome/Myalgic Encephalomyelitis and Multiple Sclerosis
 http://www.hindawi.com/journals/jir/2016/9064529/
"HIV disrupts proper CD8+ T-cell signaling"
 http://www.ncbi.nlm.nih.gov/pubmed/15053338

Thought for the Day, February 18, 2016


Wednesday, February 17, 2016

We're in the middle of an HHV-6 pandemic. Why isn't it being considered as the possible underlying infection in Zika-related disease?

http://www.statnews.com/2016/02/17/zika-dengue-infections/

It could not be simpler.


Is the Flavivirus called Clasical Swine Fever Virus the real Zika culprit in Colombia?

Are Pig diseases in Colombia causing human disease?

"In the last decade, there has been a serious increase in Colombia’s pig production. Its pig inventory almost doubled from 2.5 million in 2005 to 4.6 million in 2015. In the most pig dense province Antioquia the pig population almost quadrupled: from 465,000 in 2005 to 1.6 million in 2015."

http://www.pigprogress.net/Growing-Finishing/General/2016/2/Colombias-pig-industry-faces-disease-import-challenges-2750334W/

Classical Swine Fever Outbreaks Hit Colombia



http://www.thepigsite.com/swinenews/39133/classical-swine-fever-outbreaks-hit-colombia/

Or is this Pig Disease related to the Zika epidemic?

Animal health situation

The first outbreak of PEDv in Colombia took place in March 2014. Since then there has been a steady increase in the number of new cases. Between December 2014 and January 2015 there was unofficial information about a possible case of PEDv in the most pig-dense province of Colombia, Antioquia. Because of this the Colombian Agricultural Institute (ICA) and Colombia’s Pig Producers Association (Asoporcicultores) started a monitoring system based on offering free diagnosis for suspicious cases of PEDv. Their measures included arranging quarantine and transport control for the detected positive cases of PEDv.

 http://www.pigprogress.net/Growing-Finishing/General/2016/2/Colombias-pig-industry-faces-disease-import-challenges-2750334W/

More than 5,000 pregnant women in Colombia have Zika virus: government

 http://www.foxnews.com/health/2016/02/15/more-than-5000-pregnant-women-in-colombia-have-zika-virus-government.html

Monday, February 15, 2016

If Zika Virus variants are found in Brazil's pigs, will the data be shared?

http://www.nature.com/articles/nmicrobiol201628

The truth is gonna be huge!

The Fauci Policy on telling the public the truth about HHV-6 and the Chronic Fatigue Syndrome epidemic:

 

With friends like these . . .

How Dan Peterson's Friend Mary Guinan Helped Corrupt the Hard Drive of AIDS and Chronic Fatigue Syndrome Research

     Historians who want to trace the series of missteps that led to the HHV-6 pandemic and Holocaust II may benefit from taking a close look at a little known researcher at the CDC who played a curious role in both of the supposedly separate AIDS and chronic fatigue syndrome epidemics. Her surprising inability to see an obvious link between the two syndromes may be one of the important seeds of the whole HHV-6 disaster. She is mentioned in both the Shilts history of the early AIDS epidemic and Hillary Johnson’s journalistic account of the CDC’s bungling of the epidemic of facetiously-labeled chronic fatigue syndrome.
     According to Shilts, Mary Guinan worked for James Curran in the CDC’s venereal disease division. She was the person who sent James Curran the first ill-fated report on the first cases of what would eventually be called “AIDS” in “homosexuals.” With fellow VD chasers Harrold Jaffee and Curran, she shared the CDC AIDS Task Force’s  preoccupation with venereal diseases epidemiology. She helped impose the CDC heterosexist venereal groupthink on the emerging data of what would eventually be gayified epidemiologically into “Gay Related Immunodeficiency (GRID).”
     Ironically, considering what turned out to be the role of HHV-6 in AIDS, Shilts reported that in 1981, “on a hunch, Guinan called a drug company that manufactured medicine for severe herpes infections. They told her about a New York City doctor who had been seeing . . . dreadful herpes infections in gay men.” (ATBPO p.72) Shilts wrote that “Guinan was shaken by her investigation. She was accustomed to dealing with venereal diseases, ailments for which you receive an injection and are cured. This was different. She couldn’t get the idea out of her head: There’s something out there that’s killing people. That was when Mary Guinan hoped against hope that they would find something environmental to link these cases together. God help us, she thought, if there’s a new contagion spreading such death.” (ATBPO p.72) One way that God certainly wasn’t helping was by having a VD-obsessed doctor and her colleagues trying to comprehend a pandemic that wasn’t, strictly speaking, venereal.
     In Shilts’s account of Guinan, seeing the epidemic through gay-obsessed lenses was a given. He wrote about one of her days in 1981: “It had been another typical day of gay cancer studies for Mary Guinan. She had wakened at 6 a.m to breakfast with gay doctors and community leaders and asked again and again, ‘What’s new in the community?’ What new element might have sparked this catastrophe.” (ATBPO p.82) It was just gay, gay, gay—24/7—for the AIDS Task Force. They simply couldn’t wash the gay out of their hair. It was one of those times when every gay person should have checked to see whether they still had their wallets. Someone was about to sell them a gay epidemiological bridge.
     As Shilts sympathetically presents Guinan, he inadvertently nails the whole CDC psychological and sociological bias problem: “Guinan felt helpless and frightened. This was the meanest disease she had ever encountered. She strained to consider every possible nuance of these peoples’ lives.” (ATBPO p.83) What she really meant was gay nuances of gay lives. It is supremely ironic that Shilts wrote, “The CDC, she knew, needed to work every hypothesis imaginable into the case-control study.” (ATBPO p.83) Every hypothesis imaginable? Not by a long shot. How about the hypothesis that these cases were just extreme versions of the DuBois 1980 Atlanta cases that the CDC had been informed about? The un-gay cases.
     The process of identifying the emergence of the epidemic in nongay drug users, as described in Shilts’s book, makes it clear how gaycentric the thinking of the pioneers of the AIDS epidemiological paradigm was: “At the CDC there was a reluctance to believe that intravenous drug users might be wrapped into the epidemic, and the New York physicians also seemed obsessed with the gay angle, Guinan thought. ‘He’s said he’s not homosexual but he must be,’ doctors would confide in her.” (ATBPO p.83) Everybody was becoming an expert on gayness in those days. Given the reluctance to even see connections in those cases of nongay drug-using outcasts, it should come as no surprise when years later anyone saw the obvious connections between the epidemics of AIDS and chronic fatigue syndrome they were treated like they were strictly out to lunch. The AIDS paradigm was fatefully and messily intertwined with all the psychological baggage of sexual titillation and repulsion. If the CDC was unprepared psychologically to see drug users “wrapped into the epidemic,” how about all the good clean living white heterosexuals with the AIDS-like permutations of the immune system that characterize chronic fatigue syndrome?
     Guinan’s San Francisco trip with Harold Jaffe to interview AIDS patients and heterosexual controls also revealed the CDC mindset: “The CDC staffers could tell gay from straight controls by the way they reacted to the questions about every aspect of their intimate sexual lives. Heterosexuals seemed offended at queries about the preferred sexual techniques, while gay interviewees chatted endlessly about them.” (ATBPO p.96) Oh those gays! A herd of chatty Cathies if ever there was one. Given the bias-laden epidemiology that this chattiness was about to imprison the gay community in, one is tempted to say that loose gay lips sank a proverbial legion of gay ships. If one were watching this on a screen in a movie theater, one would want to scream out to the clueless gay interviewees for their own sake, “For Heaven’s sake, shut up!”
     Guinan was one of the CDC researchers credited by Shilts with recognizing that hemophiliacs and blood transfusion recipients might ultimately also become victims of “gay pneumonia.” She also was one of the first to worry about the AIDS infection possibilities of “semen depositors.” (ATBPO p.132) Guinan cast a wide net: “No sooner had she convinced the CDC that intravenous drug users were indeed a category of GRID cases separate from gay men, then her field of investigations discovered the first reported GRID cases among prisoners and prostitutes.” (ATBPO p.132) Unfortunately epidemiological net wasn’t wide enough to catch the 1980 DuBois Atlanta Cases of immune dysfunction. Also unfortunately for her, she helped create the very consequential epidemiological urban myth of Patient Zero. She was the first person to come in contact with Gaetan Dugas the so-called gay Typhoid Mary who the CDC would turn into the “Patient Zero” or more appropriately, “Scapegoat Zero,” of the epidemic depending on your point of view. He would become an icon for all the venereal gaycentric thinking down at the CDC.
     In one of those amazing moments in Holocaust II in which a scientist comes so face-to-face with the truth but fails to see what is right before their eyes, Shilts reports that when Guinan was studying drug users, “blood sampling of the intravenous drug users also revealed that, although many were infected with cytomegalovirus, the viral strains were all different. This was strong evidence that the herpes virus had not developed some new virulent strain. No single strain emerged, lending further weight to Don Francis’s hypothesis that a new virus, not CMV was a work.” (ATBPO p.133) The CDC, in retrospect, was most likely eyeballing strains of an undiscovered virus that would be called HBLV when Gallo’s scientists supposedly “discovered” it in 1986. It was subsequently named HHV-6. In retrospect it is pretty obvious that the CDC was looking at HHV-6 but thinking it was only CMV. (And those who wanted to see a retrovirus would have been especially predisposed not to see a new DNA virus like HBLV/HHV-6.)
     It is interesting and perhaps revealing that Guinan and her colleagues could deal with the fact that the disease or syndrome manifested itself differently in gay men and drug users—presumably for reasons that would ultimately be figured out. But God forbid that anyone would subsequently suggest that even though there were differences in the manifestations of chronic fatigue syndrome and AIDS that they were essentially manifestations of the same agent and the same pandemic. Distinctions were not turned into differences where drug users and gays were concerned, but where the gays with AIDS and the middle-class straights with chronic fatigue syndrome were concerned, every distinction,—even the teeny-tiniest or most irrelevant kind—was immediately considered a dramatic how-dare-you-compare-these-apples-and-oranges difference. Such bogus thinking would be at the heart of the “chronic fatigue syndrome is not AIDS” paradigm which would guide public health through the next three decades.
     For all her good work Guinan was eventually rewarded with the position of assistant CDC director. Unfortunately for all the victims of HHV-6, what she did do at the CDC didn’t have as much impact on the well being of the world as what she did not do. It was Guinan in 1985 who got a call from Dan Peterson, a former colleague and one of the two doctors who are credited with recognizing an outbreak of the absurdly named “chronic fatigue syndrome” in their Lake Tahoe practice. According to Hillary Johnson, “The two had become friends during a shared stint at the at the University of Utah hospital in Salt lake City in 1976.” (OW p.31) Also, according to Johnson, “Peterson had frequently sought her counsel on different infectious disease cases; he had also struck her as a gifted diagnostician.’ (OW. P.31)
     Johnson reported that “Guinan listened as her former colleague described his Tahoe patients, her curiosity aroused by the possibility that this ailment, which three recent medical papers had described, was occurring in epidemic form. Previously, researchers had described it as a sporadic illness. She remembered too, that Atlanta clinician Richard DuBois had made a presentation to agency staff on the malady early in 1983, even proposing that the new mono-like syndrome might be a second epidemic of immune dysfunction rising concurrently with AIDS.” (OW p.31)
     Did this lead Guinan serendipitously into a more complicated epidemiological vision of a variable epidemic that included both what was called “AIDS” and “chronic fatigue syndrome”? Not on your life. These first CFS patients were not gay and not drug users. They were from medical practices that could be described as being devoted to folks who ride in the middle and front part of society’s bus. Such stark social differences would make it of no consequence or interest that study after study would show one immunological and neurological similarity after another between AIDS and chronic fatigue syndrome. Guinan had helped build a paradigm that was so gay, gay, gay and so socially radioactive that the links between AIDS and CFS would be willfully ignored, buried alive by denial, and through a kind of determined public health radio silence, for all intents and purposes, be covered-up big time.
     Ignoring the obvious, Guinan sent the future “CFS” patients of America on one of the greatest medical wild goose chases in history. According to Johnson, she passed the Peterson cases on to Larry Shonberger, chief of the CDC’s epidemiology within the Division of Viral and Ricketsial Diseases. Not surprisingly, Johnson reports that “Schonberger and his staff of epidemiologists had a mandate to monitor and occasionally investigate outbreaks of viral diseases, with the exception of AIDS, which by 1985 had been awarded a separate division and staff and more than half of the federal agency’s entire annual research budget.” (OW p.32) And so, because of Guinan’s phone call and her very questionable judgment, CFS research headed down exactly the wrong road
     Had Guinan wisely directed the Lake Tahoe cases in the direction of the CDC’s AIDS division back in 1985, there was still a chance that the political and medical apartheid of the “chronic fatigue syndrome is not AIDS’ paradigm and Holocaust II might not have been able to fully materialize. But AIDS had been so gayified and turned into such a sexual bogeyman and scarlet letter syndrome, that Guinan and everyone else at the CDC couldn’t for the life of them admit that average (i.e. heterosexual) Americans were coming down with any similar or related form of acquired immunodeficiency. Instead those people were given the whitewash of a diagnosis of chronic fatigue syndrome. Those good country people, to borrow a term from Flannery O’Connor, couldn’t in a million years be suffering from something that had at one time or another been called Gay Cancer, Gay Pneumonia, and Gay Related Immunodeficiency. After all, they weren’t gay.

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