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Saturday, January 19, 2019

How Harvard's Anthony Komaroff betrayed the Chronic Fatigue Syndrome community.

An excerpt from The Chronic Fatigue Syndrome Epidemic Cover-up.

     As the story of so-called chronic fatigue syndrome unfolded in the pages of the Native, an unfortunate pattern emerged in which today’s research heroes often became tomorrow’s backsliders. Neenyah Ostrom wrote about one of the first depressing developments in the March 12 issue, the transformation of Anthony Komaroff. Ostrom asked, “What is happening to one of the country’s premiere chronic fatigue syndrome (CIDS) researchers, Dr. Anthony Komaroff? Komaroff, who is affiliated with Harvard Medical School and the prestigious Brigham and Women’s Hospital in Boston, has accomplished what the Centers for Disease Control have still been unable to do: estimate the incidence of CIDS. Komaroff and co-workers did a landmark study published in 1987 that estimated that as much as 21 percent of the general population displays symptoms of CIDS. Komaroff also has vigorously opposed the ‘psychoneurotic’ theory of CIDS that is perpetrated by such researchers as Stephen Straus at the National Institute of Allergy and Infectious Diseases.”  

     Ostrom worried that Komaroff seemed to be backtracking, insofar as his patients were being given handouts (as of September, 1989) about CFS that stated, “There is no evidence that CFS seriously damages the immune system.” Ostrom noted, “In fact, there is a great deal of evidence that [chronic fatigue syndrome] damages the immune system, either by over stimulation (resulting in production of allergies and other sensitivities) or suppression (such as the development of anergy, or complete lack of response to antigens). Komaroff, himself, along with his co-workers, has dismissed some of that evidence.” He was part of a very disturbing trend in which CFS researchers (most likely because the empirical research always seemed to inconveniently point CFS in the direction of AIDS) talked out of both sides of their mouths. Ostrom wrote, “Now, however, Komaroff seems to be leaning towards accepting the theory that CIDS is not a communicable illness, but a syndrome that is produced by an idiosyncratic response to stress or certain environmental agents. He cannot entirely get around the fact that there is immune system involvement in [chronic fatigue syndrome], however, and he mentions that ‘new viruses that affect the immune system’ might someday be implicated in causing [chronic fatigue syndrome]. But he rushes to reiterate that the immune system is ‘not severely weakened.’ Reading the new Komaroff discussing possible causes of [chronic fatigue syndrome] is rather like watching a man play a game of ping pong by himself.” One could say the epidemic of AIDS/CFS consisted of the whole biomedical establishment of America playing the same game with itself. 

     Ostrom speculated that the reason for the shift in Kamaroff’s perspective may have come from his desire to “allay the fears of his patients. In his handout he wrote, ‘You may have heard that there are some immunological problems with CFS. Many people have been frightened by this. We have been studying this question with immunological colleagues. There is no evidence that CFS seriously damages the immune system, or prevents the body from being able to fight off serious infections.’ ”  
     In essence, he was saying, “Don’t worry your pretty little heads about CFS possibly being a form of AIDS. Move along, there’s nothing alarming to see here.” Unfortunately, several of the other pioneers of CFS research would soon join his disheartening revisionism in one way or another.
     In her piece, Ostrom asked a question that could have been asked every day for the next three decades: “Wouldn’t [chronic fatigue syndrome] patients—many of whom read the scientific literature on the illness assiduously—be better served by being told the truth? Sugar pills and phony psychologizing have not yet ‘cured’ people with [chronic fatigue syndrome]; perhaps recognition that their damaged immune systems need to be bolstered would lead to improved health.” Ostrom even went out on a limb and asked, “Is there in fact an active cover-up of the breadth and severity of CIDS being conducted by the United States health authorities? And is participating in that cover-up the price individual research teams must pay to be recipients of federally funded research grants?”

HHV-6 is discussed in this Chronic Fatigue Syndrome Amazon bestseller on pages 98, 100, 122, 133-135, 143-147, 150, 153, 159, 163, 166, 179, 183, 192, 193, 198, 199, 200, 202, 204, 205, and 211.

Ampligen, the breakthrough treatment for Chronic Fatigue Syndrome is discussed in the Amazon bestseller on pages 192, 193, 202, 203, 268, 269, 360, 362, 387, 391, 410, 413, 420, and 421.

Anthony Fauci, the architect of the CFS cover-up, is discussed in this Amazon bestseller on pages 71, 103, 104, 26, 130-133, 139, 143, 157, 161, 169-171, 173, 174, 221, 254, 289, 292, 293, 300, 320-322, 324, 335, 356-358, 361, 367, and 398.

Dr. Paul Cheney, one of the honest pioneers of Chronic Fatigue Syndrome research is discussed in the Amazon bestseller on pages 145, 146, 155, 166, 192, 219, 225, 226, 232, 242, 244, 326, 327, 352, 402, 434, and 435.

Konnie Knox's pioneering research into HHV-6 is discussed in this book on page 428-431.

Hillary Johnson's brilliant reporting on Chronic Fatigue Syndrome is discussed in this Amazon bestseller on pages 407, 424, 431, 435, 436, 438 and 449.

The CDC's organized lying about the nature of  the AIDS epidemic is discussed in this book on page 432.

The role of pharmaceutical companies in developing deadly propaganda about AIDS is discussed in this book on page 440.

The crimson crescents in the throats of patients with Chronic Fatigue Syndrome are discussed in this book on page 443. 

The link between Chronic Fatigue Syndrome and Gulf War Syndrome is discussed in this book on page 447.

The crooked science of Robert Gallo is discussed on pages 31-51, 53, 54, 59, 78, 79, 93-107, 121, 126, 128, 131, 133, 134, 143-147, 152, 171, 179, 181, 101, 192, 198-200, 213, 214, 221-224.

The transmission of Chronic Fatigue Syndrome between people and their pets is discussed in this Amazon bestseller on pages 198, 225, 226, 242, 243, and 323. NK cells in CFS are discussed in this book on pages 158, 159, 160, 170, 183, 192, 200, 203, 204, 214, 242, 278, 279, 280, 282, 319, 323, 364, 370, 391, 398, 422.

On April 16, 1996, Congressman Jerrold Nadler spoke on the floor of Congress about his request for a General Accounting investigation into how the CDC had handled the Chronic Fatigue Syndrome epidemic. Nadler did that at the urging of Charles Ortleb, the publisher and the New York Native and his reporter Neenyah Ostrom. Ortleb and Ostrom had made the case to Nadler that Chronic Fatigue Syndrome and the virus it had been linked to, HHV-6, were serious public health issues.         
In an interview in New York Native with Neenyah Ostrom,Congressman Nadler said, "Congress can mandate research into CFS as a viral disease. Maybe it will turn out that HHV-6A is the cause of CFS; maybe it will turn out that other viruses are involved. But Congress can mandate research into CFS as a contagious, viral disease. I will certainly try to get Congress to do that as soon as possible."

Unfortunately, back in 1996, Nadler's warning to Congress and the medical establishment fell on deaf ears. But now that the Democrats have regained power in the House of Representatives, the newly prominent Congressman Nadler may finally be able to bring the Chronic Fatigue Syndrome epidemic and HHV-6 to the public's attention.

This book by Charles Ortleb, which details Neenyah Ostrom's diligent reporting on Chronic Fatigue Syndrome, is necessary reading for anyone who wants to know the whole history of an epidemic which has been hidden in plain sight. For a decade, starting in 1988, Ostrom reported on Chronic Fatigue Syndrome and the damage that the virus HHV-6 does to patients. What her reporting uncovered about the true nature of the Chronic Fatigue Syndrome epidemic will shock you. 

In The Chronic Fatigue Syndrome Epidemic Cover-up, Charles Ortleb recounts his newspaper's fascinating struggle to get the medical and political establishment to pay attention to Ostrom's pioneering investigative reporting on Chronic Fatigue Syndrome. 

By the time you finish Ortleb's stunning memoir, you will understand why the CDC has been unwilling to tell the public the truth about Chronic Fatigue Syndrome. The CDC does not want the public to know that Chronic Fatigue Syndrome is a transmissible illness linked to a virus that affects every system in the body. They have covered up the illness for so many decades that the neglected virus is totally out of control. Now it is causing a long list of other illnesses and many cancers. The CDC has put us all in danger.

Ostrom's decade of reporting on HHV-6 was recently vindicated by this statement from scientists at the University of Wurzburg:"While HHV-6 was long believed to have no negative impact on human health, scientists today increasingly suspect the virus of causing various diseases such as multiple sclerosis or chronic fatigue syndrome. Recent studies evensuggest that HHV-6 might play a role in the pathogenesis of several diseases of the central nervous system such as schizophrenia, bipolar disorder, depression or Alzheimer's." 

The big question about Neenyah Ostrom and New York Native is this: How many lives would have been saved if the scientific establishment and the mainstream media had paid more attention to Neenyah Ostrom's reporting on HHV-6 and Chronic Fatigue Syndrome in New York Native?             

One day, if there is any justice in the world, the CDC and the medical establishment will apologize for not paying attention to Neenyah Ostrom's groundbreaking work on Chronic Fatigue Syndrome that Charles Ortleb published in New York Native. That would be a fitting end to one of journalism's greatest David and Goliath stories.    

Anyone who wants to help Congressman Nadler and the other members of Congress who are trying to end the suffering of millions of people with Chronic Fatigue Syndrome, needs to read The Chronic Fatigue Syndrome Epidemic Cover-up.

Spotify podcasts about the HHV-6 and Chronic Fatigue Syndrome cover-up 

Anyone concerned about Kaposi's sarcoma in Chronic Fatigue Syndrome should read this excerpt from The Chronic Fatigue Syndrome Epidemic Cover-up

     In the same issue, Neenyah Ostrom reported on a new develop-ment that should have given pause to anyone married to the HIV/AIDS paradigm. She wrote, “In a striking reversal of ‘AIDS’ dogma, two recent scientific reports presented data demonstrating that the Human Immunodeficiency virus (HIV) is not the cause of Kaposi’s sarcoma (KS). These reports, in the British medical journal The Lancet, suggest that the KS seen in gay men (and others) with ‘AIDS’ may be caused by an ‘as yet unidentified’ infectious, sexually transmitted agent.”

     In another ill-considered judgment, the AIDS establishment, rather than recognizing this development as an epidemiological warning from nature that HIV-negative KS meant that HIV could not be the cause of AIDS (since KS was considered one of the dramatic hallmarks of AIDS), decided to split HIV/AIDS and KS into separate epidemics rather than lose face with the public by admitting that they had gotten the basics of the epidemic dead wrong.

     Ostrom pointed out that neither of the new Lancet reports “takes into account research performed in 1985 by researchers at the University of Miami School of Medicine in which KS was found in greater than 94 percent of autopsied patients with ‘AIDS,’ leading those investigators to postulate that ‘this autopsy series suggests that Kaposi’s sarcoma may be present in all patients with AIDS.’ (L.B. Moskovitz et al., Human Pathology, May, 1985).”

     In their study, the University of Miami scientists stated that their “findings indicated that Kaposi’s sarcoma is more common and has a wider morphological spectrum in AIDS than is generally appre-ciated.”

     Ostrom also reported, “More than 90 percent of the patients studied by the group displayed ‘microscopic evidence of Kaposi’s sarcoma in one or more organs.’ In fact, these investigators found that ‘in only one of the patients was Kaposi’s sarcoma limited to the skin’; only 26 percent of the group had cutaneous (on the surface of the skin) KS. The most common sites of identification of KS were lymph nodes and spleen.”

     For anyone with half a brain, what the University of Miami research suggested was that KS was the fundamental pathological event in AIDS, not a secondary one, and the presence of it in people without HIV indicated that HIV could possibly be ruled out as the real cause of AIDS. One of the reasons this did not register with the scientific community was that rampant fear among doctors had resulted in a limited number of autopsies being performed on AIDS patients. More autopsies might have resulted in a shift in the whole HIV/AIDS paradigm.

     The University of Miami scientists stated, “The [94.2%] prevalence of Kaposi’s sarcoma in patients with AIDS that was observed in this series had not been reported previously; there are a number of possible explanations for this disparity.” Ostrom reported that one of the reasons for the discrepancies was “that the autopsies examined many organs not just skin and occasional lymph nodes. They note, however, that autopsy ‘is not an infallible method’ for identifying KS. For example, tissues can be altered beyond recognition by other infectious agents; inadequate sampling (of lymph node tissue, primarily) can lead to a missed identification; and in some instances, autolysis, the spontaneous disintegration of tissues after death, can occur.”

     The most chilling conclusion of their study, was one that should have been another dramatic beginning of the end of the HIV theory of AIDS: “It is possible that, as we suspect, all of the patients in this series had Kaposi’s sarcoma, although we could recognize it in only 94 percent. . . . The remarkable occurrence of Kaposi’s sarcoma in T-cell domains in virtually all of our cases suggests it may play a more important role in the pathogenesis of AIDS than is generally appreciated. We believe that Kaposi’s sarcoma contributes to the deterioration of cellular immunity seen in patients with AIDS by invasive destruction of T-cell domains, as in lymphoma or Hodgkin’s disease.”

     Katie Leishman also reported on the story, in the January 28 issue of the Los Angeles Times, and Ostrom covered her piece: “The CDC explains these results, Leishman states, by postulating that a second epidemic must have started at the same time and in the same populations as the ‘AIDS’ epidemic. According to the CDC, HIV is still responsible for ‘everything except Kaposi’s sarcoma.’ ”

     Leishman wrote, in the L.A. Times, “One is driven to wonder whether the researchers might have been correct on the point they have abandoned—that Kaposi’s sarcoma and all the other dismaying symptoms of AIDS do indeed have the same cause—but are wrong in the one that they still cling to: that HIV is the cause of AIDS. To question this has been denounced as heresy. But it was also once heresy to question HIV’s role in Kaposi’s sarcoma.”

     Even Randy Shilts, one of the fiercest acolytes of the HIV para-digm, was shaken by the findings. According to Leishman’s report he said, “It is the strangest twist in terms of medical news in the epidemic in years. It calls into question everything—the existing paradigm for the epidemic, the direction of research treatment modalities, and even the integrity of the blood supply.”

     What this major development made us wonder about, at the Native, was whether there was an epidemic of unrecognized Kaposi’s sarcoma spreading throughout America and the rest of the world, an epidemic of a form of KS that was perhaps slower and different in its manifestations, perhaps mostly internal and not obvious on the skin. Was variable, chronic KS the real AIDS epidemic? We immediately wondered if chronic fatigue syndrome patients (who officially were HIV-negative) had a form of KS in their internal organs that nobody would even think to look for. Or dare to. 

Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up.

"Persons infected with KSHV can asymptomatically shed the virus. It is advised to practice safe sex with infected individuals and curtail activities where saliva might be shared during sexual activity."

This is what oral Kaposi's Sarcoma looks like.


Compare it to these crimson crescent lesions in the mouths of Chronic Fatigue Syndrome patients.

"Burke A. Cunha, MD, discovered what he called crimson crescents in the mouths of 80% of his CFS patients. After the word got out, Cunha received calls from other parts of the country. Physicians began telling him that they also were finding the crimson crescents in their patients once they looked for them."

Chronic Fatigue Syndrome patients may have undiagnosed internal Kaposi's Sarcoma. Susan Levine found HHV-8, the Kaposi's Sarcoma virus, in half of CFS patients she looked at.

Prevalence in the Cerebrospinal Fluid of the Following Infectious Agents in a Cohort of 12 CFS Subjects

Susan Levine

Published online: 04 Dec 2011

Over the last decade a wide variety of infectious agents has been associated with the chronic fatigue syndrome (CFS) as potential etiologies for this disorder by researchers from all over the world. Many of these agents are neurotrophic and have been linked previously to other diseases involving the central nervous system (CNS). Human herpes virus-6 (HHV-6), especially the B variant, has been found in autopsy specimens of patients who suffered from multiple sclerosis. Because patients with CFS manifest a wide range of symptoms involving the CNS as shown by abnormalities on brain MRIs, SPECT scans of the brain and results of tilt table testing we sought to determine the prevalence of HHV-6, HHV-8, Epstein-Barr virus (EBV), cytomegalovirus (CMV), Mycoplasma species, Chlamydia species, and Coxsackie virus in the spinal fluid of a group of 12 patients with CFS. Although we intended to search mainly for evidence of actively replicating HHV-6, a virus that has been associated by several researchers with this disorder, we found evidence of HHV-8, Chlamydia species, CMV and Coxsackie virus in 6/12 samples. Attempts were made to correlate the clinical presentations of each of these patients, especially the neurological exams and results of objective testing of the CNS, with the particular infectious agent isolated. It was also surprising to obtain such a relatively high yield of infectious agents on cell free specimens of spinal fluid that had not been centrifuged. Future research in spinal fluid analysis, in addition to testing tissue samples by polymerase chain reaction (PCR) and other direct viral isolation techniques will be important in characterizing subpopulations of CFS patients, especially those with involvement of the CNS.

Everything you wanted to know about Kaposi’s Sarcoma in Chronic Fatigue Syndrome patients and the growing CFS epidemic of HHV-8, one of the two or three viruses that may be causing Kaposi’s Sarcoma.

Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up, a bestseller on Amazon.

     Neenyah Ostrom began one of my favorite series of articles in the same issue. Titled “The Color Purple,” Ostrom reported, “Burke Cunha, M.D. who is chief of infectious disease at Winthrop-University Hospital (Mineola, Long Island), has described what he calls ‘crimson crescents’ that appear in the throats of more than 80 percent of chronic fatigue syndrome (CFS) patients. Cunha describes the crescents not only as ‘crimson,’ but ‘purplish.’ The reddish-purplish regions found in CFS patients’ throats sounded quite similar to KS (Kaposi’s sarcoma) in the throat, commented an ‘AIDS’ doctor [who wished to remain anonymous] to whom they were described. Is it possible that the crimson crescents observed in the throats of CFS patients are actually a type of KS?”      Ostrom raised the possibility that the lesions in the throats of CFS patients connected them to the theory that Florida researchers held about KS being the unrecognized but unifying central pathological event AIDS. As I previously reported, the Florida team, headed by Dr. George Hensley, had turned the AIDS paradigm upside down, by finding KS in nearly 100% of AIDS patients, when they explored the internal organs closely during autopsies of AIDS patients. Their fascinating work suggested that KS preceded AIDS and caused more of the immune problem in AIDS than previously thought.

     Basically, Ostrom was asking if the KS-like lesions, in the tonsils of [CFS]patients, were an indication that some kind of unrecognized indolent KS was present internally, something that physicians would not even be thinking about because of the conceptual wall that socially hostile epidemiology had built between AIDS and chronic fatigue syndrome. And the CFS patients were not particularly interested in finding out if they shared KS with AIDS patients.

    Ostrom went even further, in the July 20 issue, and speculated that the dramatic digestive problems in chronic fatigue syndrome were actually the result of the unrecognized chronic or slowly progressive KS in the CFS patients’ digestive tracts. Ostrom noted that Dr. Carol Jessop, who was talking to a group of patients at a chronic fatigue syndrome conference, said, “Almost all patients would say to me, ‘I was totally well until I got this [chronic fatigue syndrome],’ and yet, when I took their past medical histories, I found it wasn’t quite true. Now these aren’t disastrous problems. In fact, if they had gone to their physicians for any of these problems such as irritable bowel, diarrhea and constipation, abdominal cramping, bloating, flatulence, chronic constipation, heartburn, etc., their physician would probably just say, ‘Oh, take this’ and that would be it. So we as physicians didn’t relate to our patients that this was a problem, so they considered themselves to be totally healthy. Yet, if you look at the numbers, 89 percent of the [chronic fatigue syndrome] patients had irritable bowel syndrome, diarrhea alternating with constipation, and abdominal cramping pain episodically. Another 80 percent complained of constant gas, bloating and flatulence. It’s amazing that we can all meet in this room together.”       Ostrom wondered if “Jessop may have uncovered a fallacy in the prevailing wisdom of chronic fatigue syndrome: that it begins as a respiratory, flu-like illness. Instead, as she points out, it may be a digestive tract disturbance. Jessop’s statistic—that more than 80 percent of CFS patients complain of irritable bowel syndrome, abdominal pain, gas, bloating, etc.—corresponds to the more than 80 percent of CFS patients who exhibit a red-to-purplish crescent-shaped lesion in their throats. (Helot, Paul, in the New York Times Long Island edition, January 14, 1992) . . . What if the digestive problems described by the CFS patients are actually caused by KS in the gastrointestinal tract? According to the AIDS Treatment News, ‘The most common HIV-related causes of gastric symptoms include KS, lymphoma, and CMV [cytomegalovirus].’ And while KS is unusual in the esophagus, it ‘may occasionally be found there.’ KS also can cause colitis and diarrhea . . . in people with AIDS.” Ostrom noted, “Gastrointestinal symptoms, it is realized in retrospect, were among the first signs of the ‘AIDS’ epidemic; and, it now seems, were also among the first symptoms seen in the CFS epidemic. That observation raises what should be a relatively simple question to answer: Are the gastrointestinal symptoms in both patient populations caused, in part, by undetected KS?”

Excerpted from The Chronic Fatigue Syndrome Epidemic Cover-up, a bestseller on Amazon.

Important information about the Kaposi’s Sarcoma problem in Chronic Fatigue Syndrome

Whatever happened to the concern about controlling the Kaposi's Sarcoma Virus? What about all the infected Chronic Fatigue Syndrome patients?

Is Kaposi's Sarcoma responsible for the digestive disorders in Chronic Fatigue Syndrome?

HHV-8 is a Kaposi's Sarcoma cancer virus in many AIDS and Chronic Fatigue Syndrome patients and is spread by kissing but the CDC couldn't care less.

Company founded by Robert Gallo suggests 65% of gay men are infected with Kaposi's Sarcoma virus.

Coagulation issues may link Chronic Fatigue Syndrome, Kaposi's Sarcoma, and AIDS

Should Chronic Fatigue Syndrome be added to the spectrum of Kaposi's Sarcoma-Associated Herpesvirus, or Human Herpesvirus 8, Diseases?

Why Susan Levine may have done the world's most important research on Chronic Fatigue Syndrome.

Does HHV-8 viral load raise questions about the legitimacy of HIV viral load?

Can Chronic Fatigue Syndrome patients with internal Kaposi's Sarcoma pass it on to their partners?

Can most of the symptoms of Chronic Fatigue Syndrome described by Paul Cheney be attributed to internal Kaposi's Sarcoma?

Is Chronic Fatigue Syndrome Associated Kaposi's Sarcoma  (CFSKS) a diagnosis all doctors should become aware of?

Stanford University and Open Medicine Foundation should have a conference on diagnosing Kaposi's Sarcoma in Chronic Fatigue Syndrome.

Why are doctors not looking for Kaposi's Sarcoma in Chronic Fatigue Syndrome patients?

If Chronic Fatigue Syndrome involves HHV-8 and Kaposi's Sarcoma, scientists will have to ask if it came from pigs.

Does the Red Blood Cell Deformability Issue Link Chronic Fatigue Syndrome to Kaposi's Sarcoma and AIDS?

Will the Montoya cytokine study show that Chronic Fatigue Syndrome is Kaposi's Sarcoma Inflammatory Syndrome?

Is Chronic Fatigue Syndrome a Kaposi's Sarcoma inflammatory cytokine syndrome?

How Kaposi's Sarcoma almost undermined the HIV theory of AIDS

How did 50% of Chronic Fatigue Syndrome patients become infected with a Kaposi’s Sarcoma cancer virus?

Has the moment finally come to address the issue of Kaposi's Sarcoma in Chronic Fatigue Syndrome?

Oral Kaposi's Sarcoma looks like the Crimson Crescents in Chronic Fatigue Syndrome patients.

Were oral crimson crescents the first obvious sign of Kaposi's Sarcoma in Chronic Fatigue Syndrome patients?

Did Paul Cheney ever consider the possibility that Chronic Fatigue Syndrome patients have internal Kaposi's Sarcoma?

Is the red blood cell deformability issue another clue that Chronic Fatigue Syndrome is also a Kaposi's Sarcoma Syndrome?

Why is nobody warned about exposure to HHV-8, the Kaposi's Sarcoma virus that even patients with Chronic Fatigue Syndrome are sometimes infected with?

Do petechiae in Chronic Fatigue Syndrome connect it to Kaposi's Sarcoma, HHV-8, and AIDS?

Whatever is causing Kaposi's Sarcoma may be the real cause of Chronic Fatigue Syndrome and AIDS.

A massive epidemic of Kaposi's Sarcoma may be coming.

When Kaposi's Sarcoma almost turned AIDS upside down.

Human herpesvirus 6 activates lytic cycle replication of Kaposi's sarcoma-associated herpesvirus.

All AIDS patients have some form of Kaposi's Sarcoma in this study. Is the same true for Chronic Fatigue Syndrome?

Crimson crescents may suggest that all Chronic Fatigue Syndrome patients have Kaposi's Sarcoma.

Do all Chronic Fatigue Syndrome patients have an indolent form of Kaposi's Sarcoma?

Are these marks on the skin a sign of Kaposi’s Sarcoma in Chronic Fatigue Syndrome?

On autopsy, do the inflamed ganglia of Chronic Fatigue Syndrome patients resemble Kaposi's Sarcoma?

What people don't know about Kaposi's Sarcoma in Chronic Fatigue Syndrome and AIDS.

Do all Chronic Fatigue Syndrome patients show internal Kaposi's Sarcoma upon autopsy?

Decades ago, a New York newspaper sounded the alarm about Kaposi’s Sarcoma in Chronic Fatigue Syndrome. The book about that newspaper is now a must-read bestseller on Amazon. Purchase a hardcover, paperback, or Kindle version here.

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