Why isn't China testing its pigs in Wuhan and Hubei for SARS-CoV-2? This experiment is beating around the bush.

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"Growing the virus in animal cells is one way to test whether the pathogen has adapted to a new host. Shi Yi, a microbiologist at the CAS Institute of Microbiology in Beijing, plans to introduce an inactivated version of RATG13, in various animals, such as bats, cats monkeys and pigs, and see whether the virus develops a similar pattern of mutations to SARS-CoV-2 over time. If similarities emerge, that could reveal which animals the virus adapted to before it jumped to people. "

https://www.nature.com/articles/d41586-020-01449-8

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Anna Fogdell Hahn, researcher at the Department of Clinical Neuroscience at Karolinska Institutet in Sweden. MS linked to variant of HHV-6 (the CFS/AIDS virus) through new method Researchers at Karolinska Institutet have developed a new method to separate between two different types of a common herpes virus (HHV-6) that has been linked to multiple sclerosis. By analyzing antibodies in the blood against the most divergent proteins of herpesvirus 6A and 6B, the researchers were able to show that MS-patients carry the herpesvirus 6A to a greater extent than healthy individuals. The findings, published in Frontiers in Immunology, point to a role for HHV-6A in the development of MS. 
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Is reactivation of HHV-6 major problem in Covid-19?

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  " Dursun  et al .  noticed an increased rate of pityriasis rosea and Kawasaki disease in patients who presented to dermatology outpatient clinics during the COVID‐19 pandemic.  The authors attributed that to coronavirus triggering of HHV‐6 (i.e. endogenous viral reactivation of HHV‐6 by SARS‐CoV‐2 infection)." https://onlinelibrary.wiley.com/doi/10.1111/ijd.15215
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WTF?

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Science and Society at the Crossroads: Skepticism vs. Denial and Elitism vs. Public Engagement Report on an international meeting at Chicheley Hall, 15-16 June 2015 Principal organizer and curator: Prof Stephan Lewandowsky (University of Bristol) https://figshare.com/articles/RS_scienceandsociety_September_2015_pdf/2061696
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How corrupt is the CDC?

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 Read this Amazon.com: The Four Horsemen of the Chronic Fatigue Syndrome Apocalypse: How the Bigotry and Incompetence of Four AIDS Scientists at the CDC Helped Create the Chronic Fatigue Syndrome Disaster eBook : Ortleb, Charles: Kindle Store
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Gallo's early statements about HHV-6

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Gallo's early statements about HHV-6 are discussed in this excerpt from Nick Regush's The Virus Within (page. 13).   When NCI laboratory research revealed the extent of HHV-6's destructive power, Robert Gallo began to speak out about the implications. On May 10, 1988, at the annual meeting of the American Society of Microbiology in Miami, he told an audience that HHV-6 had been found along with HIV in T-4 cells, and speculated that the new herpes virus could be a "co-factor" in AIDS. One month later, at an international AIDS conference in Stockholm, he explained that HHV-6 killed T-4 cells in the test tube more effectively than did HIV, and said the new herpes virus could cause massive cell death. Gallo reasoned that HHV-6 infection might help speed up the HIV-led breakdown of the immune system in AIDS, although he cautioned that NCI research on HHV-6 was still preliminary. The Virus Within: A Coming Epidemic: Regush, Nicholas: 9780525945345: Amazon.com: Books
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Support the International HHV-6 Protest in Berlin July 23-26, 2017

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The International HHV-6 Protest  and Teach-in in Berlin  During the HHV-6 Foundation's 10th International  Conference on HHV-6 & 7 July 23-26, 2017 Overview Thousands of civil rights activists and public health activists should meet at Freie Universitat Berlin during the HHV-6 Foundation's International Conference on HHV-6 & 7 (July 23-26) to start a new HHV-6 public health and civil rights movement. They should demand reparations for everyone who has been harmed by the HIV Fraud Ponzi Scheme and the HHV-6 Cover-up. They should also demand that key players in the HIV Fraud Ponzi Scheme and HHV-6 Cover-up be removed from positions of power and science and medicine : Robert Gallo, Luc Montagnier, Anthony Fauci, Dharam Ablashi, Anthony Komaroff, Henri Agut, Dharam Ablashi, Myron Essex and Phil Pellet.   Freie Universitat Berlinshould consider cancelling classes for three days for a HHV-6 Teach-in. HHV-6 will ...
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Need background on HHV-6? Start with these books:

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The Virus Within by Nicholas Regush (Print Edition) What Really Killed Gilda Radner? Frontline Reports on the Chronic Fatigue Syndrome Epidemic by Neenyah Ostrom (Print Edition) 50 Things You Should Know About the Chronic Fatigue Syndrome Epidemic by Neenyah Ostrom (Print Edition) America's Biggest Cover-Up: 50 More Things Everyone Should Know About the Chronic Fatigue Syndrome Epidemic And Its Link to AIDS by Neenyah Ostrom (Print Edition) The Closing Argument by Charles Ortleb (Available on Kindle and in a Print Edition. Free for Kindle owners with Amazon Prime.)
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Has Maureen Hanson found another opportunistic infection that links Chronic Fatigue Syndrome and AIDS?

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  The Enterovirus Theory of Disease Etiology in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Critical Review Adam J. O'Neal  and  Maureen R. Hanson * Abstract Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) is a complex, multi-system disease whose etiological basis has not been established. Enteroviruses (EVs) as a cause of ME/CFS have sometimes been proposed, as they are known agents of acute respiratory and gastrointestinal infections that may persist in secondary infection sites, including the central nervous system, muscle, and heart. To date, the body of research that has investigated enterovirus infections in relation to ME/CFS supports an increased prevalence of chronic or persistent enteroviral infections in ME/CFS patient cohorts than in healthy individuals. Nevertheless, inconsistent results have fueled a decline in related studies over the past two decades. This review covers the aspects of ME/CFS pathophysiology that are consistent with a ...
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Bhupesh Prusty, Konnie Knox, and Brigitte Huber have done the most important research on HHV-6. Why is Huber's retroviral finding about HHV-6 being ignored????

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  HHV-6A infection induces expression of HERV-K18-encoded superantigen Albert K. Taia, Janos Lukab, Dharam Ablashi, Brigitte T. Huber published online 08 June 2009. Abstract Background The human endogenous retrovirus K-18 (HERV-K18) encodes a superantigen that causes deregulation of the immune system. This provirus is transcriptionally silent, but can be induced by Epstein–Barr virus (EBV) infection and IFN-α treatment. Objectives Since the herpesvirus EBV induces HERV-K18 expression in human B cells, it was of interest to determine if other herpesviruses would have similar HERV-K18 transactivation properties. Human herpesvirus (HHV)-6A, a neurotropic virus associated with multiple sclerosis, was a logical candidate for these studies. Study design HSB2 cells (HHV-6-negative control), HSB2-ML cells (containing latent HHV-6A genome) and HSB2/HHV-6A cells (HSB-2 cells productively infected with HHV-6A) were compared for their level of HERV-K18 transcription, using quantitative RT...
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Groq on miRNAs and retroviruses.

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 Are miRNAs ever confused with retroviruses? Yes, microRNAs (miRNAs) and retroviruses can be confused with each other due to some similarities in their structure and function. Here are a few reasons why: Both miRNAs and retroviruses are small RNA molecules: miRNAs are typically 18-25 nucleotides in length, while retroviruses can have varying lengths, but are often around 5-10 kilobases. However, some retroviruses, such as human T-cell leukemia virus type 1 (HTLV-1), have shorter RNA genomes that are similar in size to miRNAs. Both can integrate into host genomes: miRNAs can integrate into host genomes through a process called RNA-directed DNA methylation, while retroviruses can integrate their genomes into host DNA through a process called reverse transcription. Both can regulate gene expression: miRNAs regulate gene expression by binding to messenger RNA (mRNA) and preventing its translation, while retroviruses can regulate gene expression by inserting their genomes into host DNA ...
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https://hhv-6foundation.org/cihhv-6/the-activation-of-cihhv-6-by-telomeric-circle-formation-qa-with-dr-bhupesh-prusty Tweet If you have Amazon Prime or Kindle Unlimited, you can immediately begin reading The Chronic Fatigue Syndrome Epidemic Cover-up and you will soon understand why the facts about the Chronic Fatigue Syndrome epidemic have been hidden from the public for almost four decades. Click here to begin reading Or purchase a print copy for only $20 shipped by Amazon Prime  If you want to know the truth about the Chronic Fatigue Syndrome epidemic, you need to discover the reporting of Neenyah Ostrom. For a decade, starting in 1988, Ostrom reported on Chronic Fatigue Syndrome for a newspaper called New York Native . What her reporting uncovered about the true nature of the Chronic Fatigue Syndrome epidemic will shock you.                                 ...
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